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Inhibition of beta-glucuronidase in human urine by ascorbic acid
J C Young1, E M Kenyon, E J Calabrese
1Environmental Health Sciences Program, School of Public Health, University of Massachusetts, Amherst 01003.
Human & Experimental Toxicology
|May 1, 1990
Abstract:
The effects of oral supplementation of ascorbic acid (1500 mg d-1) on urinary beta-glucuronidase (beta-G) activity was assessed in a double-blind crossover study design over a 3-week period. The subjects on ascorbic acid treatment displayed a statistically significant (P less than 0.05) decrease in beta-G with an approximate decrease of 25%.