Related Experiment Video

Updated: May 9, 2026

Establishment of Acute Pontine Infarction in Rats by Electrical Stimulation
05:02

Establishment of Acute Pontine Infarction in Rats by Electrical Stimulation

Published on: August 27, 2020

Pure motor trigeminal neuropathy in a woman with tegmental pontine infarction

Dae-Hyun Kim1, Jong Kuk Kim, Ji-Yoon Kang

  • 1Department of Neurology, Dong-A University College of Medicine, 1, 3-ga, Dongdaesing-dong, Seo-gu, Busan 602-715, Republic of Korea; Busan-Ulsan Regional Cardiocerebrovascular Center, Medical Science Research Center, Dong-A University College of Medicine, Busan, Republic of Korea.

Journal of Clinical Neuroscience : Official Journal of the Neurosurgical Society of Australasia
|July 10, 2013
PubMed

A 50-year-old woman presented with drowsiness, severe dysarthria, left facial palsy, and left hemiparesis. Diffusion-weighted MRI showed cerebral infarction in the left pontine tegmentum and right cerebellum and angiography revealed basilar artery occlusion. Altered consciousness and hemiparesis fully resolved following successful recananlisation of the basilar artery by emergent intra-arterial thrombolysis. However, about 8 months later, obvious wasting of the left temporal and buccal area without sensory signs was detected. Electrophysiological evaluation revealed a chronic denervation process in the left masseter and temporalis muscles. We suggest that a small infarction in the trigeminal nucleus of the pontine tegmentum can cause an isolated trigeminal motor neuropathy with masticatory muscle atrophy.

Keywords:
Isolated trigeminal motor neuropathyMasseter musclePontine infarction

More Related Videos

In-Vivo Calcium Imaging of Sensory Neurons in the Rat Trigeminal Ganglion04:39

In-Vivo Calcium Imaging of Sensory Neurons in the Rat Trigeminal Ganglion

Published on: February 9, 2024

Related Concept Videos

Transient Ischemic Attack l: Introduction01:26

Transient Ischemic Attack l: Introduction

A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Cranial Nerves: Types Part I01:14

Cranial Nerves: Types Part I

Cranial nerves are responsible for transmitting motor and sensory information between the brain and various parts of the body. There are twelve pairs of cranial nerves, with the first six being essential in sensory perception, motor control, and autonomic functions related to the head and neck.
Olfactory Nerve (Cranial Nerve I)
The olfactory nerve, or cranial nerve I, is unique as it is purely sensory and dedicated to the sense of smell. This nerve originates in the olfactory epithelium of the...