Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Predator-Prey Interactions02:39

Predator-Prey Interactions

Predators consume prey for energy. Predators that acquire prey and prey that avoid predation both increase their chances of survival and reproduction (i.e., fitness). Routine predator-prey interactions elicit mutual adaptations that improve predator offenses, such as claws, teeth, and speed, as well as prey defenses, including crypsis, aposematism, and mimicry. Thus, predator-prey interactions resemble an evolutionary arms race.
Steps in Outbreak Investigation01:18

Steps in Outbreak Investigation

In the ever-evolving field of public health, statistical analysis serves as a cornerstone for understanding and managing disease outbreaks. By leveraging various statistical tools, health professionals can predict potential outbreaks, analyze ongoing situations, and devise effective responses to mitigate impact. For that to happen, there are a few possible stages of the analysis:
Pharmacodynamic Models: Direct Effect Model and Indirect Response Model01:29

Pharmacodynamic Models: Direct Effect Model and Indirect Response Model

Pharmacodynamic models are essential tools in understanding the relationship between drug concentrations and their effects on biological systems. By characterizing the dynamics of drug action, these models guide dose selection, optimize therapeutic efficacy, and inform the development of new drugs. Two major classes of pharmacodynamic models include direct effect and indirect response models.Direct Effect ModelsDirect effect models describe the immediate relationship between drug concentration...
Microbial Interactions: Predation01:28

Microbial Interactions: Predation

Microbial predation refers to the process by which one microorganism kills and consumes another to obtain nutrients and energy. It encompasses both bacterial and protozoan predators. This interaction plays a crucial role in shaping microbial communities and regulating nutrient cycling.Bacterial Predators: Epibiotic vs. EndobioticBacterial predators are classified based on their mode of attack as either epibiotic or endobiotic. Epibiotic predators, such as Vampirococcus, attach to the surface of...
Pharmacodynamic Models: Overview01:27

Pharmacodynamic Models: Overview

Pharmacodynamic (PD) responses describe the interaction between a drug and its biological target, culminating in a physiological effect. These responses can be classified into different types: continuous variables, such as blood glucose levels; categorical outcomes, like survival rates; and time-to-event metrics, such as disease progression. Understanding and modeling PD responses are critical for optimizing drug efficacy and safety.PD models describe the relationship between drug concentration...
Pharmacodynamic Models: Additive and Proportional Drug Effect Model01:09

Pharmacodynamic Models: Additive and Proportional Drug Effect Model

Drug response models describe how pharmacological agents interact with biological systems to produce measurable effects. Baseline responses are inherent physiological activities without a drug significantly influencing the observed pharmacological outcomes. Depending on the drug response model employed, these baseline responses may combine with the drug's effect in either an additive or proportional manner.Additive Drug Response ModelIn the additive model, the drug effect is independent of the...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Cofactor functions of magnesium in complement activation and antimicrobial responses.

Expert review of clinical immunology·2026
Same author

Population-Based First Estimates of the Effect of HPV Vaccination in Three Italian Areas Covered by Organised Cervical Screening.

International journal of cancer·2026
Same author

Bifurcation, chaos, multistability, and pattern formation of a discrete predator-prey model with Allee effect.

Chaos (Woodbury, N.Y.)·2026
Same author

A genome-wide association study identifies EYA2 as a contributing gene for diabetic retinopathy in type 2 diabetes.

Communications medicine·2026
Same author

A Stochastic Dynamical Model for Sympatric Speciation in a Two-phenotype Population.

Theoretical biology forum·2026
Same author

The Histopathological Spectrum of Breast Lesions in Bangladeshi Women.

Cureus·2026

Related Experiment Video

Updated: May 9, 2026

A Rat Model of EcoHIV Brain Infection
08:48

A Rat Model of EcoHIV Brain Infection

Published on: January 21, 2021

Comparing functional responses in predator-infected eco-epidemics models.

Mainul Haque1, Md Sabiar Rahman, Ezio Venturino

  • 1Division of Anaesthesia & Intensive Care, School of Clinical Sciences, Queen's Medical Centre, University of Nottingham, UK.

Bio Systems
|August 3, 2013
PubMed
Summary

This study models predator-prey dynamics with disease in predators, finding that infection rate and competition significantly impact population stability and survival. Key parameters determine ecosystem behavior, including species persistence and oscillations.

Keywords:
Biological implicationsIntra-specific competition among the predatorLimit cyclesPredator–prey modelsTransmissible disease in predator

More Related Videos

A Mouse Model for the Transition of Streptococcus pneumoniae from Colonizer to Pathogen upon Viral Co-Infection Recapitulates Age-Exacerbated Illness
12:21

A Mouse Model for the Transition of Streptococcus pneumoniae from Colonizer to Pathogen upon Viral Co-Infection Recapitulates Age-Exacerbated Illness

Published on: September 28, 2022

Using a Bacterial Pathogen to Probe for Cellular and Organismic-level Host Responses
08:38

Using a Bacterial Pathogen to Probe for Cellular and Organismic-level Host Responses

Published on: February 22, 2019

Related Experiment Videos

Last Updated: May 9, 2026

A Rat Model of EcoHIV Brain Infection
08:48

A Rat Model of EcoHIV Brain Infection

Published on: January 21, 2021

A Mouse Model for the Transition of Streptococcus pneumoniae from Colonizer to Pathogen upon Viral Co-Infection Recapitulates Age-Exacerbated Illness
12:21

A Mouse Model for the Transition of Streptococcus pneumoniae from Colonizer to Pathogen upon Viral Co-Infection Recapitulates Age-Exacerbated Illness

Published on: September 28, 2022

Using a Bacterial Pathogen to Probe for Cellular and Organismic-level Host Responses
08:38

Using a Bacterial Pathogen to Probe for Cellular and Organismic-level Host Responses

Published on: February 22, 2019

Area of Science:

  • Mathematical Biology
  • Ecology
  • Epidemiology

Background:

  • Predator-prey models are fundamental in ecology.
  • Incorporating disease dynamics into these models is crucial for realistic ecological understanding.
  • Transmissible diseases can significantly alter population dynamics.

Purpose of the Study:

  • To analyze mathematical models of predator-prey systems with disease affecting only the predator population.
  • To investigate the influence of various predator functional responses on eco-epidemic dynamics.
  • To establish the existence, boundedness, and uniqueness of solutions for the proposed models.

Main Methods:

  • Development and analysis of four distinct mathematical models.
  • Application of stability and bifurcation analysis.
  • Investigation of predator functional responses, including attack rates and competition.

Main Results:

  • Existence, boundedness, and uniqueness of solutions were mathematically established.
  • Stability analysis revealed critical dependencies on key parameters.
  • Bifurcation analysis indicated potential shifts in population dynamics.

Conclusions:

  • Eco-epidemic predator-prey system behavior, including stability, extinction, and oscillations, is critically dependent on the rate of infection, predator interspecies competition, and attack rates.
  • Functional responses play a significant role in shaping eco-epidemic outcomes.
  • The study provides insights into the biological implications of mathematical modeling in disease ecology.