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Published on: May 13, 2019
Constrictive pericarditis developed after childhood repair of ventricular septal defect
Hirofumi Saiki1, Satoshi Masutani, Masanori Tamura
1Department of Pediatrics and Pediatric Cardiology, Saitama Medical University, Saitama, Japan.
Insights
Constrictive pericarditis can be a serious delayed complication after congenital heart surgery. Echocardiography is crucial for diagnosing this condition and guiding treatment for improved heart failure outcomes.
Area of Science:
- Cardiology
- Cardiac Surgery
- Medical Imaging
Background:
- A patient with a history of surgical closure of ventricular septal defect (VSD) presented with syncope.
- The patient experienced a long-term uneventful postoperative course before hospitalization.
Observation:
- Diagnosis of atrial flutter and echocardiographic findings of poor ventricular motion and dilated atria.
- Despite atrial flutter termination, persistent heart failure symptoms and limited improvement were noted.
- Computed tomography revealed localized calcification, insufficient to explain severe diastolic dysfunction.
Findings:
- Mitral annular tissue Doppler imaging waveform strongly suggested constrictive pericarditis.
- Pericardial decortication led to significant improvement in heart failure symptoms.
Implications:
- Constrictive pericarditis is a potential life-threatening complication years after congenital heart surgery.
- Functional assessment using echocardiography is vital for establishing the relationship between imaging findings and clinical presentation.
- Early and accurate diagnosis of constrictive pericarditis is essential for effective management in post-VSD repair patients.
Abstract:
The patient was an 18-year-old man who had undergone surgical closure of ventricular septal defect at the age of 2 years. The patient was urgently hospitalized due to syncope after longstanding uneventful postoperative course, and diagnosed with atrial flutter. Echocardiography showed poor ventricular motion and markedly dilated both atriums. After termination of atrial flutter, ventricular motion improved remarkably; however, there was little improvement in heart failure. Although we suspected constrictive pericarditis, calcification observed on computed tomography was too localized to fully explain the cause of severe diastolic dysfunction. Based on the waveform of mitral annular tissue Doppler imaging, we were strongly convinced of the underlying physiology of constrictive pericarditis. Pericardial decortications markedly improved heart failure symptoms. This case emphasizes that constrictive pericarditis could be a life-threatening complication in the remote phase after congenital heart surgery. It also emphasizes the importance of functional assessment by echocardiography to identify the cause-effect relation between imaging and clinical symptoms.
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