Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Transient Ischemic Attack l: Introduction01:26

Transient Ischemic Attack l: Introduction

35
A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
35
Aneurysm II: Clinical Manifestations and Diagnostic Studies01:21

Aneurysm II: Clinical Manifestations and Diagnostic Studies

593
Thoracic, aortic arch and abdominal aneurysms are significant vascular conditions that can present with various clinical manifestations and lead to serious complications. Understanding these manifestations and the appropriate diagnostic studies is essential for effective management and treatment.Thoracic Aortic AneurysmsThoracic aortic aneurysms often remain asymptomatic until they reach a size that impinges on adjacent structures. They typically cause deep, diffuse chest pain that radiates to...
593
Atherosclerosis II: Clinical Manifestations and Diagnostic Tests01:27

Atherosclerosis II: Clinical Manifestations and Diagnostic Tests

961
Atherosclerosis is a progressive disorder that leads to the thickening and narrowing of arterial walls due to plaque buildup. This condition can cause various symptoms depending on the arteries affected:Coronary Artery Disease (CAD): This condition affects the coronary arteries and may lead to chest pain (angina), shortness of breath (dyspnea), heart attacks, and other heart disease symptoms.Cerebrovascular Disease: This affects blood flow to the brain, causing transient ischemic attacks (TIAs)...
961
The Arch of Aorta01:10

The Arch of Aorta

2.2K
The coronary arteries, originating from the ascending aorta, bifurcate from two sinuses located within the ascending aorta. Positioned just above the aortic semilunar valve, these sinuses house essential aortic baroreceptors and chemoreceptors, crucial for maintaining cardiac function. The left coronary artery and the right coronary artery branch off from the left posterior and anterior aortic sinuses, respectively.
Encircling the heart, the coronary arteries form a ring-like structure before...
2.2K
Aneurysm I: Introduction01:30

Aneurysm I: Introduction

660
An aortic aneurysm is a localized outpouching or dilation at a weak point in the artery wall. It may involve different parts of the aorta, such as the abdominal aorta, aortic arch, or thoracic aorta.Etiological factorsSeveral disorders are associated with aortic aneurysms.Congenital causes, such as primary connective tissue disorders like Marfan syndrome, impact the integrity and strength of connective tissues, notably affecting the aorta. Marfan syndrome is a genetic disorder that specifically...
660
Aortic Regurgitation II: Clinical Features and Diagnostic Tests01:22

Aortic Regurgitation II: Clinical Features and Diagnostic Tests

1.2K
Aortic valve regurgitation (AR) occurs when the aortic valve fails to close properly, allowing blood to flow backward from the aorta into the left ventricle. This backflow can result in two distinct clinical presentations: acute and chronic AR, each characterized by its own set of symptoms and physical findings.Acute Aortic RegurgitationAcute AR presents with a sudden onset of severe symptoms. Patients typically experience profound dyspnea (shortness of breath), chest pain, and signs of left...
1.2K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Tick-borne anaplasmosis and myocarditis in eastern Ontario.

CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne·2026
Same author

Myocardial Calcification as a Rare Cardiac Sequela: When Sepsis Leaves Its Mark.

JACC. Case reports·2026
Same author

The value of preoperative risk scores prior to tricuspid valve interventions: The TRI-SCORE and beyond.

Annals of cardiothoracic surgery·2026
Same author

Management of patients with severe tricuspid regurgitation: A proposed decision algorithm guided by the TRI-SCORE.

The Journal of thoracic and cardiovascular surgery·2026
Same author

Global adoption of TRI-SCORE digital tools for the risk stratification of patients with tricuspid regurgitation.

Archives of cardiovascular diseases·2026
Same author

Objective Evaluation of Congestion in Patients With Tricuspid Regurgitation.

The Canadian journal of cardiology·2026

Related Experiment Video

Updated: May 7, 2026

Isolation and Analysis of Aortic Arch and Root Lesions in an Atherosclerotic Mouse Model
07:49

Isolation and Analysis of Aortic Arch and Root Lesions in an Atherosclerotic Mouse Model

Published on: February 14, 2025

2.8K

Aortic arch atheroma in transient ischemic attack patients.

Céline Guidoux1, Mikael Mazighi, Philippa Lavallée

  • 1Department of Neurology and Stroke Centre, Bichat University Hospital, INSERM U-698 and Paris-Diderot University, Paris, France.

Atherosclerosis
|October 16, 2013
PubMed
Summary

Aortic arch atheroma (AAA) affects 40% of transient ischemic attack (TIA) patients and is linked to recurrent vascular events. Identifying AAA in TIA patients is crucial for better risk assessment.

Keywords:
AortaAtherosclerosisPlaqueStroke

More Related Videos

Author Spotlight: Assessing Ischemic Stroke Damage Through Middle Cerebral Artery Occlusion Model
05:32

Author Spotlight: Assessing Ischemic Stroke Damage Through Middle Cerebral Artery Occlusion Model

Published on: August 11, 2023

3.2K
A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia
06:01

A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia

Published on: August 18, 2015

14.4K

Related Experiment Videos

Last Updated: May 7, 2026

Isolation and Analysis of Aortic Arch and Root Lesions in an Atherosclerotic Mouse Model
07:49

Isolation and Analysis of Aortic Arch and Root Lesions in an Atherosclerotic Mouse Model

Published on: February 14, 2025

2.8K
Author Spotlight: Assessing Ischemic Stroke Damage Through Middle Cerebral Artery Occlusion Model
05:32

Author Spotlight: Assessing Ischemic Stroke Damage Through Middle Cerebral Artery Occlusion Model

Published on: August 11, 2023

3.2K
A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia
06:01

A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia

Published on: August 18, 2015

14.4K

Area of Science:

  • Cardiology
  • Neurology
  • Vascular Medicine

Background:

  • Aortic arch atheroma (AAA) is a known risk factor for stroke.
  • The prevalence and prognostic significance of AAA in patients experiencing transient ischemic attack (TIA) are not well-defined.

Purpose of the Study:

  • To determine the prevalence of aortic arch atheroma (AAA) in patients with transient ischemic attack (TIA).
  • To investigate the association between AAA and stroke risk factors.
  • To evaluate the prognostic impact of AAA on recurrent vascular events in TIA patients.

Main Methods:

  • Utilized data from the SOS-TIA registry, including 1231 patients who underwent transesophageal echocardiography (TEE).
  • Assessed AAA prevalence, grading it as moderate (<4 mm) or severe (≥4 mm).
  • Followed patients for one year to monitor recurrent vascular events.

Main Results:

  • Overall AAA prevalence was 40% (26% moderate, 14% severe) among TIA patients.
  • AAA was independently associated with intracranial or extracranial stenosis (≥50%).
  • Recurrent vascular event rates increased with AAA severity (2.2% without AAA, 4.1% moderate AAA, 6.6% severe AAA).

Conclusions:

  • Systematic identification of AAA in TIA patients is recommended.
  • AAA presence and severity aid in optimizing risk stratification for TIA patients.
  • Further research may clarify the prognostic role of AAA in TIA management.