Related Experiment Video

Updated: May 6, 2026

Surgical Technique for the Implantation of a Biomimetic Artificial Intervertebral Disc in a Goat Animal Model
07:06

Surgical Technique for the Implantation of a Biomimetic Artificial Intervertebral Disc in a Goat Animal Model

Published on: October 10, 2025

820

Acute hind limb paralysis secondary to an extradural spinal cord Cryptococcus gattii lesion in a dog

Lindsey Kurach1, Chris Wojnarowicz, Tom Wilkinson

  • 1Michigan State University, College of Veterinary Medicine, East Lansing, Michigan, USA (Kurach); Prairie Diagnostic Services, University of Saskatchewan, Saskatoon, Saskatchewan (Wojnarowicz); Washington State University, College of Veterinary Medicine, Pullman, Washington, USA (Wilkinson); Guardian Veterinary Centre, Edmonton, Alberta (Sereda).

The Canadian Veterinary Journal = La Revue Veterinaire Canadienne
|October 25, 2013
PubMed

More Related Videos

Intraoperative Ultrasound in Spinal Surgery05:53

Intraoperative Ultrasound in Spinal Surgery

Published on: August 17, 2022

4.9K
Longitudinal Evaluation of Mouse Hind Limb Bone Loss After Spinal Cord Injury using Novel, in vivo, Methodology10:39

Longitudinal Evaluation of Mouse Hind Limb Bone Loss After Spinal Cord Injury using Novel, in vivo, Methodology

Published on: December 7, 2011

16.4K

Related Concept Videos

Secondary Spinal Cord Injury llI: Pathophysiology01:25

Secondary Spinal Cord Injury llI: Pathophysiology

Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...
72
Brain Abscess l: Introduction01:26

Brain Abscess l: Introduction

A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial...
48