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Human tumor xenografts treated with recombinant human tumor necrosis factor alone or in combination with interferons

Cancer Research
|August 1, 1986
PubMed

Insights

Recombinant human tumor necrosis factor (rHuTNF) showed significant antitumor activity in mouse xenografts when administered intratumorally. Intraperitoneal administration was less effective, but could be enhanced with interferons.

Area of Science:

  • Oncology
  • Immunology
  • Pharmacology

Background:

  • Recombinant human tumor necrosis factor (rHuTNF) is a cytokine with potential antitumor properties.
  • Understanding the efficacy of rHuTNF against various human tumors is crucial for therapeutic development.

Purpose of the Study:

  • To evaluate the antitumor activity of rHuTNF in human tumor xenografts in nude mice.
  • To compare the efficacy of intratumoral versus intraperitoneal administration of rHuTNF.
  • To assess the potential synergistic effects of rHuTNF with interferons.

Main Methods:

  • Six human tumor xenograft lines (breast and bowel) were established in nude mice.
  • Recombinant human tumor necrosis factor (rHuTNF) was administered daily intratumorally (5 µg) or intraperitoneally (5 µg or 25 µg).
  • Tumor growth, regression, and histological changes were monitored; inflammatory cell infiltration was assessed.

Main Results:

  • Intratumoral rHuTNF led to complete regression in 3/6 xenografts and stasis/slowing in 2/6 within 3-4 weeks.
  • Histology showed tumor cell degeneration and inflammatory cell infiltration post-treatment.
  • Intraperitoneal rHuTNF showed limited efficacy, with some growth slowing in 2/6 lines at 5 µg.
  • Higher intraperitoneal doses (25 µg) showed marginal improvement; co-administration with interferons enhanced efficacy.

Conclusions:

  • Intratumoral administration of rHuTNF is significantly more effective than intraperitoneal administration for these xenografts.
  • rHuTNF induces tumor cell degeneration and inflammatory responses.
  • Combination therapy with interferons may enhance the therapeutic potential of intraperitoneal rHuTNF.

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