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Analysis of middle tumor antigen and pp60c-src interactions in polyomavirus-transformed rat cells

Journal of Virology
|October 1, 1987
PubMed

Insights

Polyomavirus middle tumor antigen (MTAg) stably associates with pp60c-src kinase in transformed cells, influencing cellular tyrosine kinase activity. This interaction does not alter the electrophoretic mobility of pp60c-src.

Area of Science:

  • Molecular biology
  • Virology
  • Oncology

Background:

  • Polyomavirus middle tumor antigen (MTAg) is implicated in cellular transformation.
  • pp60c-src is a non-receptor tyrosine kinase involved in cell signaling and proliferation.

Purpose of the Study:

  • To quantify the association between pp60c-src and MTAg in polyomavirus-transformed cells.
  • To investigate the impact of this association on pp60c-src activity and tyrosine kinase activity associated with MTAg.

Main Methods:

  • Quantitative immunoblot analyses were employed to detect and quantify pp60c-src and Py MTAg.
  • Immunoprecipitation assays were used to determine the relative abundance of associated proteins and their enzymatic activities.

Main Results:

  • Approximately 5-10% of immunoprecipitable pp60c-src molecules stably associate with MTAg and exhibit elevated protein kinase activity.
  • 10-15% of detectable MTAg molecules are stably associated with pp60c-src.
  • pp60c-src accounts for 50-75% of MTAg-associated tyrosine kinase activity, with other kinases contributing the remainder.
  • Associated pp60c-src molecules do not show altered electrophoretic mobility compared to free pp60c-src.

Conclusions:

  • A significant fraction of pp60c-src in polyomavirus-transformed cells is stably complexed with MTAg.
  • This complex formation correlates with enhanced pp60c-src kinase activity and contributes substantially to MTAg-associated tyrosine kinase activity.
  • The interaction does not appear to modify the basic molecular characteristics of pp60c-src.

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