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Generation of macrophage variants with 5-azacytidine: selection for mannose receptor expression

S Diment1, M S Leech, P D Stahl

  • 1Department of Cell Biology and Physiology, Washington University Medical School, St. Louis, Missouri 63110.

Insights

Researchers induced mannose receptor expression in macrophage cell lines using 5-Azacytidine. This allowed the cells to perform mannose receptor-mediated functions, like endocytosis and phagocytosis.

Area of Science:

  • Immunology
  • Cell Biology

Background:

  • Primary macrophages express mannose receptors, crucial for cellular functions.
  • Macrophage-derived cell lines typically lack surface mannose receptor expression, limiting their utility.
  • Investigating methods to restore mannose receptor expression in cell lines is important for research.

Purpose of the Study:

  • To induce and sustain mannose receptor expression in a macrophage-derived cell line (J774).
  • To characterize the functional capabilities of J774 cells with restored mannose receptor expression.
  • To explore the impact of mannose receptor expression on lysosomal enzyme secretion.

Main Methods:

  • Treatment of J774 cells with 5-Azacytidine to induce gene expression.
  • Fluorescence-activated cell sorting (FACS) to isolate receptor-positive cells.
  • Cloning of cells to establish stable, receptor-expressing macrophage lines.
  • Assays to measure beta-glucuronidase endocytosis and yeast phagocytosis.
  • Quantification of beta-hexosaminidase secretion.

Main Results:

  • 5-Azacytidine successfully induced mannose receptor expression in J774 cells.
  • Sorted and cloned cells demonstrated continuous mannose receptor expression in culture.
  • Mannose receptor-positive macrophages effectively endocytosed beta-glucuronidase and phagocytosed yeast.
  • Increased mannose receptor expression correlated with reduced secretion of beta-hexosaminidase.

Conclusions:

  • Mannose receptor expression can be epigenetically induced in macrophage cell lines.
  • Restored mannose receptor expression confers functional endocytic and phagocytic capabilities.
  • Mannose receptor expression influences the secretion profile of lysosomal enzymes in macrophages.

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