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Related Concept Videos

Mitral Regurgitation I: Introduction01:20

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Mitral regurgitation is characterized by the backward circulation of blood from the left ventricle to the left atrium during systole, a phase of the cardiac cycle when the heart contracts and pumps blood out of the chambers. This abnormal flow occurs primarily due to the dysfunction of the mitral valve or its supporting structures, which include the mitral leaflets, chordae tendineae, annulus, and papillary muscles.Etiology and Mechanisms:Primary Mitral Regurgitation: This type arises from...
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Mitral Valve Stenosis (MVS) is a heart condition where the mitral valve narrows, impeding blood circulation from the left atrium to the left ventricle. The etiology and pathophysiology of this condition are multifaceted, leading to a cascade of cardiovascular complications.Causes of Mitral Valve StenosisRheumatic Heart Disease: It is the main cause of mitral valve stenosis, particularly in developing nations. This condition arises from rheumatic fever, an inflammatory illness resulting from...
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Aortic valve regurgitation (AR) occurs when the aortic valve fails to close properly, allowing blood to flow backward from the aorta into the left ventricle. This backflow can result in two distinct clinical presentations: acute and chronic AR, each characterized by its own set of symptoms and physical findings.Acute Aortic RegurgitationAcute AR presents with a sudden onset of severe symptoms. Patients typically experience profound dyspnea (shortness of breath), chest pain, and signs of left...
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Systolic Heart Failure and Compensatory MechanismsSystolic heart failure (also termed HFrEF, Heart Failure with Reduced Ejection Fraction) is the most prevalent type of heart filure. It results in a decreased volume of blood being pumped from the ventricle. The aortic arch and carotid sinuses have baroreceptors that detect reduced blood pressure, triggering the sympathetic nervous system (SNS) to release epinephrine and norepinephrine. Initially, this response aims to boost heart rate and...
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Hypertrophic cardiomyopathy, or HCM, is an autosomal dominant genetic disorder characterized by asymmetric left ventricular hypertrophy without ventricular dilation. It is more common in men and is typically diagnosed in young, athletic adults.EtiologyHCM is primarily genetic and is caused by mutations in genes encoding sarcomeric proteins. Researchers have identified over 1400 mutations across at least 11 different genes. Among these, the most frequently occurring mutations are found in the...
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Related Experiment Video

Updated: May 3, 2026

Generation and Characterization of Right Ventricular Myocardial Infarction Induced by Permanent Ligation of the Right Coronary Artery in Mice
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Generation and Characterization of Right Ventricular Myocardial Infarction Induced by Permanent Ligation of the Right Coronary Artery in Mice

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Evolution of right ventricular function post-acute ST elevation myocardial infarction.

Idan Roifman1, Mohammad I Zia, Anna Zavodni

  • 1Schulich Heart Centre, Sunnybrook Research Institute, Sunnybrook Health Science Centre, University of Toronto, Toronto, ON, Canada.

Journal of Magnetic Resonance Imaging : JMRI
|January 29, 2014
PubMed
Summary
This summary is machine-generated.

Right ventricular (RV) function improves over time after myocardial infarction (MI), particularly when initially low. Cardiovascular magnetic resonance (CMR) at 3 weeks best assesses RV injury, as electrocardiograms (EKG) may underestimate damage.

Keywords:
acute myocardial infarctioncardiac magnetic resonanceright ventricle

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Area of Science:

  • Cardiology
  • Medical Imaging

Background:

  • Right ventricular (RV) dysfunction is a significant complication following myocardial infarction (MI).
  • Understanding the temporal changes in RV function and the diagnostic accuracy of imaging modalities is crucial for patient management.

Purpose of the Study:

  • To evaluate the evolution of RV function post-MI.
  • To identify the culprit vessel associated with RV injury.
  • To compare RV injury assessment between cardiovascular magnetic resonance (CMR) and electrocardiogram (EKG).

Main Methods:

  • Thirty-one patients with ST-elevation MI (STEMI) underwent CMR at three time points.
  • RV ejection fraction (RVEF) was measured to assess RV function.
  • Culprit vessel and EKG criteria for RV myocardial infarction (RVMI) were analyzed.

Main Results:

  • RV function did not significantly change in patients with initially normal scans.
  • RVEF significantly improved from baseline to 3 weeks and 6 months in patients with initially low RVEF.
  • The left anterior descending (LAD) artery was the culprit vessel in 23% of patients with low RVEF.
  • EKG criteria identified RVMI in only 15% of patients with low RVEF.

Conclusions:

  • Cardiovascular magnetic resonance (CMR) assessment at 3 weeks post-MI is optimal for evaluating RV injury.
  • Standard EKG criteria may underestimate the extent of RV injury compared to CMR findings.