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Amino acid sequence of the alpha subunit of human leukocyte adhesion receptor Mo1 (complement receptor type 3)

M A Arnaout1, S K Gupta, M W Pierce

  • 1Renal Unit, Children's Hospital, Boston, Massachusetts.

Insights

This study reports the complete amino acid sequence of the Mo1 alpha subunit, a key human leukocyte adhesion molecule. Understanding its structure, including calcium-binding domains, is crucial for elucidating its function in immune responses.

Area of Science:

  • Molecular Biology
  • Immunology
  • Cell Biology

Background:

  • Mo1 (complement receptor type 3, CR3; CD11b/CD18) is a critical adhesion molecule on human leukocytes.
  • It is a heterodimer composed of alpha (CD11b) and beta (CD18) subunits.
  • Its adhesion-promoting function is vital for leukocyte interactions.

Purpose of the Study:

  • To determine the complete amino acid sequence of the human Mo1 alpha subunit.
  • To analyze the structural domains of the Mo1 alpha subunit.
  • To understand the structural basis for Mo1's divalent cation-dependent functions.

Main Methods:

  • Deduction of amino acid sequence from complementary DNA (cDNA).
  • Bioinformatic analysis of protein domains and homology.
  • Comparison with related integrin family members.

Main Results:

  • The human Mo1 alpha subunit comprises 1,136 amino acids.
  • Key domains identified include an extracytoplasmic region with putative Ca2+-binding sites, a transmembrane segment, and a cytoplasmic tail.
  • Significant homology was observed with other integrin alpha subunits, notably leukocyte p150,95.

Conclusions:

  • The deduced amino acid sequence provides a foundation for understanding Mo1's structure-function relationships.
  • The identified Ca2+-binding domains explain Mo1's divalent cation-dependent activities.
  • A unique N-terminal region in Mo1 alpha may contribute to specific leukocyte glycoprotein functions.

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