Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Portal Hypertension01:22

Portal Hypertension

50
Portal hypertension is an increase in blood pressure within the portal venous system. Normally, this pressure is less than 5 mmHg. It is considered clinically significant when it rises above 10 mmHg. At this threshold, complications from altered blood flow and venous congestion emerge.EtiologyPortal hypertension arises from conditions that impede blood flow through the liver. The most common cause is cirrhosis, in which chronic liver injury leads to fibrotic scarring. This fibrosis narrows or...
50
Mitral Stenosis I: Introduction01:22

Mitral Stenosis I: Introduction

1.8K
Mitral Valve Stenosis (MVS) is a heart condition where the mitral valve narrows, impeding blood circulation from the left atrium to the left ventricle. The etiology and pathophysiology of this condition are multifaceted, leading to a cascade of cardiovascular complications.Causes of Mitral Valve StenosisRheumatic Heart Disease: It is the main cause of mitral valve stenosis, particularly in developing nations. This condition arises from rheumatic fever, an inflammatory illness resulting from...
1.8K
Aortic Regurgitation I: Introduction01:15

Aortic Regurgitation I: Introduction

1.6K
IntroductionAortic regurgitation is characterized by the backward flow of blood from the aorta into the left ventricle during diastole and arises from the improper closure of the aortic valve. This condition results in left ventricular volume overload and can stem from both acute and chronic etiologies, each contributing uniquely to the disease's progression and symptomatology.Acute and Chronic CausesAcute aortic regurgitation often results from events that suddenly impair the integrity of the...
1.6K
Esophageal Varices-I: Introduction01:24

Esophageal Varices-I: Introduction

2.4K
Esophageal varices are dilated, tortuous veins which are found mainly in the submucosa of the lower esophagus but which may also appear higher up or extend into the stomach. They develop due to increased pressure in the portal venous system, often as a result of liver cirrhosis. This condition scars and damages the liver, impeding normal blood flow through the portal vein. To compensate, blood seeks alternative pathways, forming fragile new vessels (varices) in the esophagus and stomach. These...
2.4K
Blood Supply to the Digestive System01:16

Blood Supply to the Digestive System

6.6K
Splanchnic circulation refers to the network of blood vessels that supply and drain blood from the abdominal organs involved in digestion, including the stomach, liver, pancreas, intestines, and spleen. This circulation delivers essential nutrients and oxygen while removing waste products from these organs.
Blood Supply to the Digestive System: The splanchnic circulation involves three main arteries: the celiac artery (also known as the celiac trunk) and the superior and inferior mesenteric...
6.6K
Mitral Stenosis IV: Nursing Management01:27

Mitral Stenosis IV: Nursing Management

422
A comprehensive nursing assessment is essential for patients with valvular heart disease, which involves any dysfunction of the heart valves that could impact blood flow and overall heart function.Subjective Data Collection:Chief Complaint and Present Illness: Start with the patient's primary concerns, focusing on the onset, duration, and progression of cardiac symptoms such as dyspnea, fatigue, chest pain, and palpitations.Past Medical History: Collect detailed information on any previous...
422

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Guiding Principles for Physician-Modified Endografts.

Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists·2026
Same author

In-Stent Stenosis or Occlusion After Carotid Artery Stenting in Patients Treated With Anticoagulants vs Antiplatelet Therapy.

Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists·2026
Same author

Pelvic venous disease and lymphatic dysfunction: evaluating the evidence for a proposed continuum.

The Journal of cardiovascular surgery·2026
Same author

A critical analysis of the Carotid Revascularization and Medical Management for Asymptomatic Carotid Stenosis Trial (CREST-2) and the European Carotid Surgery Trial (ECST)-2.

Seminars in vascular surgery·2026
Same author

An international, multispecialty consensus document on the optimal management of patients with internal carotid artery occlusion.

Journal of vascular surgery·2026
Same author

Factors Influencing Sex Specific Outcomes after Complex Endovascular Aortic Aneurysm Repair: Insights from a National Vascular Registry.

European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery·2026

Related Experiment Video

Updated: May 2, 2026

Multimodality Diagnosis of Mesenteric Ischemia
05:07

Multimodality Diagnosis of Mesenteric Ischemia

Published on: July 21, 2023

1.6K

Mesenteric stenosis, collaterals, and compensatory blood flow.

André S van Petersen1, Jeroen J Kolkman2, Robbert Meerwaldt3

  • 1Division of Vascular Surgery, Department of Surgery, Medical Spectrum Twente, Enschede, The Netherlands; Division of Vascular Surgery, Department of Surgery, Bernhoven Hospital, Oss-Uden-Veghel, The Netherlands.

Journal of Vascular Surgery
|March 22, 2014
PubMed
Summary

Stenosis in the celiac artery (CA) or superior mesenteric artery (SMA) increases flow in the other artery, especially with collateral flow. This can mimic or overstate stenosis in unaffected mesenteric arteries.

More Related Videos

A Magnetic Resonance Imaging-based Computational Protocol for Analysis of Plaque Morphology and Hemodynamics in Patients with Carotid Artery Stenosis
09:36

A Magnetic Resonance Imaging-based Computational Protocol for Analysis of Plaque Morphology and Hemodynamics in Patients with Carotid Artery Stenosis

Published on: August 12, 2025

836
Author Spotlight: Advancing Spectral Characterization of Physiological and Malperfused Tissues
04:57

Author Spotlight: Advancing Spectral Characterization of Physiological and Malperfused Tissues

Published on: July 5, 2024

941

Related Experiment Videos

Last Updated: May 2, 2026

Multimodality Diagnosis of Mesenteric Ischemia
05:07

Multimodality Diagnosis of Mesenteric Ischemia

Published on: July 21, 2023

1.6K
A Magnetic Resonance Imaging-based Computational Protocol for Analysis of Plaque Morphology and Hemodynamics in Patients with Carotid Artery Stenosis
09:36

A Magnetic Resonance Imaging-based Computational Protocol for Analysis of Plaque Morphology and Hemodynamics in Patients with Carotid Artery Stenosis

Published on: August 12, 2025

836
Author Spotlight: Advancing Spectral Characterization of Physiological and Malperfused Tissues
04:57

Author Spotlight: Advancing Spectral Characterization of Physiological and Malperfused Tissues

Published on: July 5, 2024

941

Area of Science:

  • Vascular Surgery
  • Diagnostic Imaging
  • Hemodynamics

Background:

  • The mesenteric circulation possesses extensive collateral pathways, often masking symptoms of arterial stenosis.
  • Understanding collateral flow's impact on duplex parameters is crucial for accurate diagnosis.

Purpose of the Study:

  • To investigate how collateral flow influences duplex parameters of the celiac artery (CA) and superior mesenteric artery (SMA).

Main Methods:

  • A cohort of 228 patients with suspected chronic mesenteric syndrome was analyzed between 1999 and 2007.
  • Angiography identified mesenteric vessel stenosis and collateral flow patterns.
  • The study examined the effect of stenosis in one mesenteric artery on the other, considering collateral presence.

Main Results:

  • Stenosis in the CA led to significantly higher peak systolic velocity (PSV) and end-diastolic velocity in the non-stenotic SMA, and vice versa.
  • Increasing CA stenosis severity correlated with higher SMA PSV.
  • Collateral flow significantly impacted duplex parameters in the non-stenotic SMA, notably increasing PSV with CA stenosis.

Conclusions:

  • Stenosis in either the CA or SMA elevates flow velocities in the contralateral mesenteric artery.
  • This velocity increase is linked to the presence of collateral circulation.
  • Collaterals and unilateral stenosis can lead to misinterpretation or overestimation of stenosis in the companion mesenteric artery.