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Preproenkephalin mRNA expression in developing rat heart and in cultured ventricular cardiac muscle cells

J P Springhorn1, W C Claycomb

  • 1Department of Biochemistry and Molecular Biology, Louisiana State University Medical Center, New Orleans 70112.

The Biochemical Journal
|February 15, 1989
PubMed

Insights

Heart muscle cells express preproenkephalin (ppEnk) mRNA, with levels changing during development and adulthood. Cyclic AMP and other compounds affect ppEnk mRNA levels in cultured heart cells.

Area of Science:

  • Cardiovascular Biology
  • Molecular Cardiology
  • Neuroendocrinology

Background:

  • Heart muscle tissue exhibits the highest preproenkephalin (ppEnk) mRNA levels in adult rats.
  • ppEnk mRNA is present in ventricular cardiac muscle cells and is developmentally regulated.

Purpose of the Study:

  • To investigate the developmental regulation of ppEnk mRNA expression in rat cardiac muscle.
  • To examine the effects of various compounds on ppEnk mRNA levels in primary cardiac muscle cell cultures.
  • To explore the potential roles of enkephalin in cardiac muscle cell differentiation and function.

Main Methods:

  • Quantitative analysis of ppEnk mRNA expression in rat hearts at different developmental stages.
  • Primary cell culture of neonatal and adult rat cardiac muscle cells.
  • Treatment of cultured cells with cyclic AMP, 3-isobutyl-1-methylxanthine, and phorbol 12-myristate 13-acetate.

Main Results:

  • ppEnk mRNA expression is low in early postnatal development, decreases during the third week, and peaks in adulthood.
  • ppEnk mRNA is actively expressed in both neonatal and adult cardiac muscle cell cultures.
  • Cyclic AMP and 3-isobutyl-1-methylxanthine increase steady-state ppEnk mRNA content, while phorbol 12-myristate 13-acetate has a transient effect.

Conclusions:

  • Enkephalin may have diverse roles in neonatal cardiac muscle cell differentiation and adult heart cell function.
  • Primary cardiac muscle cell culture systems are valuable for studying ppEnk mRNA regulation in the heart.

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