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Published on: August 31, 2013
Neurotoxin-induced paralysis: a case of tick paralysis in a 2-year-old child
Olga D Taraschenko1, Karen M Powers1
1Department of Neurology, Albany Medical College, Albany, New York.
Insights
Tick paralysis, a progressive weakness caused by tick bites, can mimic other neurological conditions. Prompt diagnosis and tick removal are crucial for recovery, even in non-endemic areas.
Area of Science:
- Neurology
- Toxicology
- Dermatology
Background:
- Tick paralysis is an arthropod-transmitted disease presenting as progressive ascending weakness.
- Symptoms overlap with inflammatory neurological disorders, leading to misdiagnosis.
- Misdiagnosis can result in unnecessary treatments and prolonged hospitalization.
Observation:
- A 2-year-old girl in New York presented with rapidly progressing ascending paralysis.
- Initial investigations, including toxicology and CSF analysis, were negative.
- An engorged tick was discovered on her scalp, leading to a diagnosis of tick paralysis.
Findings:
- Tick removal resulted in rapid improvement of the patient's paralysis.
- The patient recovered without further intervention after tick removal.
- This case highlights the importance of a thorough skin examination.
Implications:
- Tick paralysis should be considered in the differential diagnosis of acute progressive weakness.
- Thorough skin examination is essential in evaluating patients with unexplained paralysis.
- Early diagnosis and intervention can prevent severe outcomes and unnecessary medical care.
Background:
Tick paralysis is an arthropod-transmitted disease causing potentially lethal progressive ascending weakness. The presenting symptoms of tick paralysis overlap those of acute inflammatory diseases of the peripheral nervous system and spinal cord; thus, the condition is often misdiagnosed, leading to unnecessary treatments and prolonged hospitalization.
Patient:
A 2-year-old girl residing in northern New York and having no history of travel to areas endemic to ticks presented with rapidly progressing ascending paralysis, hyporeflexia, and intact sensory examination. Investigation included blood and serum toxicology screens, cerebrospinal fluid analysis, and brain imaging. With all tests negative, the child's condition was initially mistaken for botulism; however, an engorged tick was later found attached to the head skin. Following tick removal, the patient's weakness promptly improved with no additional interventions.
Conclusion:
Our patient illustrates the importance of thorough skin examination in all cases of acute progressive weakness and the necessity to include tick paralysis in the differential diagnosis of paralysis, even in nonendemic areas.
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