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[Defibrination syndrome after snake bites].

A Meissner1, B Hausmann, C Linn

  • 1I. Medizinische Universitätsklinik Kiel.

Deutsche Medizinische Wochenschrift (1946)
|September 29, 1989
PubMed
Summary
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Two men developed defibrination syndrome after viper bites, characterized by severe bleeding and clotting factor deficiencies. Prompt administration of specific antivenom successfully normalized clotting function within 48 hours.

Area of Science:

  • Toxicology
  • Hematology
  • Herpetology

Background:

  • Viper envenomation can lead to complex hemostatic disorders.
  • Defibrination syndrome is a severe complication characterized by depletion of clotting factors.

Observation:

  • Two male patients, aged 51 and 29, presented with defibrination syndrome within two hours of Echis carinatus and Agkistrodon halys bites, respectively.
  • Clinical manifestations included oral bleeding and macrohematuria.
  • Laboratory findings revealed afibrinogenemia, prolonged thrombin time, and the presence of fibrinogen degradation products.

Findings:

  • The syndrome was directly linked to the specific viper species involved.
  • Rapid onset of severe coagulopathy was observed post-envenomation.
  • Biochemical markers indicated consumption coagulopathy consistent with defibrination.

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Implications:

  • Early recognition and diagnosis of defibrination syndrome are crucial for effective management.
  • Timely administration of species-specific antivenom is essential for reversing coagulopathy and preventing further complications.
  • This case highlights the importance of prompt medical intervention following viper bites.