Self-limiting left ventricular wall rupture following myocardial infarction: a case report and literature review
Insights
A rare complication of heart attack, left ventricular free wall rupture, can sometimes self-limit. This case shows a patient recovering with medical treatment due to spontaneous clot formation.
Area of Science:
- Cardiology
- Cardiovascular Medicine
- Medical Case Reports
Background:
- Left ventricular free wall rupture is a rare but often fatal complication of acute myocardial infarction.
- It typically leads to haemopericardium and cardiac tamponade.
Observation:
- A 62-year-old woman presented with myocardial infarction and signs of left ventricular free wall rupture.
- Coronary angiography identified critical stenosis in the right coronary artery.
- A ventriculogram showed contrast extravasation through the inferolateral wall.
Findings:
- The patient experienced a small rupture, which appeared to self-limit.
- Spontaneous closure was likely facilitated by epicardial fibrin deposits and clot formation at the rupture site.
- The patient improved with intensive medical management alone, avoiding surgery.
Implications:
- This case highlights that not all left ventricular free wall ruptures require surgical intervention.
- Conservative management may be successful in select cases with small, self-sealing ruptures.
- Understanding spontaneous healing mechanisms is crucial for optimizing treatment strategies.
Abstract:
We report the case of a 62-year-old woman presenting with symptoms and findings of myocardial infarction and a left ventricular free wall rupture. Coronary angiography revealed a critical stenosis in the middle right coronary artery. A contrast left ventriculogram revealed extravasation of contrast through the inferolateral wall of the left ventricle. Left ventricular free wall rupture is a rare complication of acute myocardial infarction, occurring in approximately 2% of cases. It is often fatal because of the development of haemopericardium and tamponade. Some patients, like the one described in this case, may present with small leaks that might close spontaneously by epicardial fibrin deposits, thus self-limiting, without requiring surgical intervention. This patient received only intense medical treatment. Indeed, blood clots at the endocardial and the epicardial site of the rupture have often been identified, suggesting protection for further rupture.
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