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Related Concept Videos

Pulmonary Tuberculosis II01:28

Pulmonary Tuberculosis II

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Tuberculosis, or TB, is a bacterial infectious disease caused by Mycobacterium tuberculosis. While its primary impact is on the lungs, leading to pulmonary tuberculosis, it can also affect various other organs, a condition referred to as extrapulmonary tuberculosis.
Here is a detailed explanation of its pathophysiology:
Transmission: The process begins when a person inhales droplet nuclei containing M. tuberculosis. These are typically released into the air when an individual with pulmonary or...
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Pulmonary Tuberculosis V01:28

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Medical management of tuberculosis (TB) patients involves a comprehensive approach that includes diagnosis, treatment, and monitoring. The specific strategies can vary depending on the type of tuberculosis (latent or active), the patient's overall health status, and other considerations.
Latent tuberculosis infection occurs when TB bacteria are present in a person's body, but are not causing illness or symptoms. It is not contagious, and preventive treatment is crucial to avoid the...
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Pulmonary Tuberculosis III01:31

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Tuberculosis (TB) is a contagious infection primarily affecting the lung parenchyma but which can also affect other body parts. TB can be classified based on disease development, presentation, and the affected anatomical site.
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Pulmonary Tuberculosis IV01:26

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Tuberculosis, more commonly referred to as TB, is an infectious disease stemming from Mycobacterium tuberculosis. While it primarily impacts the lungs, TB can also affect other body areas. Given its severity and global impact, timely and accurate diagnosis is crucial for controlling its spread and improving patient outcomes.
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Pulmonary Tuberculosis I01:29

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Tuberculosis, often called TB, is a contagious illness primarily caused by Mycobacterium tuberculosis. It mainly affects the lung parenchyma but can also impact other body parts.
Causative Organism
The primary infectious agent causing tuberculosis is Mycobacterium tuberculosis, a slow-growing, acid-fast, aerobic rod that exhibits sensitivity to heat and ultraviolet light. Instances of Mycobacterium bovis and Mycobacterium avium contributing to the development of TB infection are rare.
Mode of...
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Acute Pyelonephritis I: Introduction01:27

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Pyelonephritis is a bacterial infection that primarily affects the renal parenchyma and collecting system, including the renal pelvis, tubules, and interstitial tissue of one or both kidneys. It can be classified as either acute—a sudden, severe infection—or chronic, which refers to long-term or recurrent kidney infections.The primary cause of acute pyelonephritis (APN) is bacterial infection, with Escherichia coli accounting for approximately 70-80% of cases. Other bacteria, such...
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Analysis of 18FDG PET/CT Imaging as a Tool for Studying Mycobacterium tuberculosis Infection and Treatment in Non-human Primates
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Rapidly progressive glomerulonephritis in tuberculosis.

Rajesh Waikhom1, Dipankar Sarkar, Mahesh Bennikal

  • 1Department of Nephrology, Jawaharlal Nehru Institute of Medical Sciences, Porompat Imphal, India.

Saudi Journal of Kidney Diseases and Transplantation : an Official Publication of the Saudi Center for Organ Transplantation, Saudi Arabia
|June 28, 2014
PubMed
Summary
This summary is machine-generated.

Tuberculosis can affect the kidneys, but glomerular involvement is rare. This case shows successful treatment of miliary tuberculosis with immune complex nephritis using anti-tuberculous therapy alone, avoiding immunosuppression.

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Area of Science:

  • Nephrology
  • Infectious Diseases
  • Immunology

Background:

  • Renal tuberculosis is typically due to lympho-hematogenous spread.
  • Glomerular involvement in renal tuberculosis is uncommon.
  • Crescentic nephritis in tuberculosis presents a therapeutic challenge due to immunosuppression risks.

Observation:

  • A patient presented with miliary tuberculosis and advanced renal injury requiring renal replacement therapy.
  • Diagnosis was confirmed by positive sputum acid-fast bacilli (AFB), caseating granulomas in lymph nodes, and positive urinary mycobacterial PCR.
  • The patient had immune complex crescentic nephritis.

Findings:

  • Complete recovery of renal function was achieved with anti-tuberculous therapy.
  • Immunosuppressive therapy was not required for managing the nephritis.
  • This suggests a potential for non-immunosuppressive treatment in select cases.

Implications:

  • This case highlights that renal tuberculosis with immune complex crescentic nephritis can be managed effectively with anti-tuberculous therapy alone.
  • It offers a less risky therapeutic approach, avoiding immunosuppression in patients with active tuberculosis.
  • Further research may explore the role of anti-tuberculous therapy as a primary treatment for tuberculous nephritis.