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Interaction of the polyene antibiotic amphotericin B with model membranes: differences between small and large

J Milhaud1, M A Hartmann, J Bolard

  • 1Laboratoire de Physique et Chimie Biomoléculaire (CNRS UA198), Université Pierre-et-Marie-Curie, Paris, France.

Biochimie
|January 1, 1989
PubMed

Insights

Large unilamellar vesicles (LUVs) provide a better model for studying amphotericin B (AmB) interactions. AmB binding differs significantly between saturated (DMPC) and unsaturated (EPC) lipid chains in LUVs, impacting membrane permeability.

Area of Science:

  • Biochemistry
  • Membrane Biophysics
  • Pharmacology

Background:

  • Amphotericin B (AmB) is a polyene antibiotic used to treat fungal infections.
  • Previous studies on AmB-membrane interactions utilized small unilamellar vesicles (SUV), which are less representative of biological membranes than large unilamellar vesicles (LUV).

Purpose of the Study:

  • To investigate the interaction of AmB with LUVs composed of different lipid compositions.
  • To compare AmB binding and membrane permeabilization in LUVs versus SUVs.
  • To elucidate the role of sterols in modulating AmB-membrane interactions.

Main Methods:

  • Circular dichroism (CD) spectroscopy to monitor AmB-lipid interactions.
  • Carboxyfluorescein (CF) release assay to assess membrane permeability.
  • Utilized dimyristoyl phosphatidyl choline (DMPC) and egg phosphatidyl choline (EPC) LUVs.

Main Results:

  • AmB exhibited strong, non-saturable binding to DMPC LUVs, independent of temperature.
  • AmB binding to EPC LUVs showed saturation at lower AmB/lipid ratios compared to DMPC LUVs.
  • Ergosterol incorporation into EPC LUVs altered CD spectral features, suggesting specific AmB-sterol complex formation and potential channel formation.
  • Significant CF release was observed from DMPC LUVs at lower AmB concentrations than from EPC LUVs.

Conclusions:

  • LUVs offer a more biologically relevant model for studying AmB interactions.
  • The lipid acyl chain unsaturation significantly influences AmB binding and membrane disruption.
  • Sterols, particularly ergosterol, play a crucial role in modulating AmB's interaction with membranes and its ability to form pores.

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