Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Graves' Disease I: Introduction01:28

Graves' Disease I: Introduction

19
Graves' disease is an autoimmune disorder that causes hyperthyroidism, or overactivity of the thyroid gland. It results from autoantibodies called thyroid-stimulating immunoglobulins (TSIs), which bind to thyroid-stimulating hormone (TSH) receptors, leading to overstimulation of hormone production and a hypermetabolic state.EtiologyAlthough considered idiopathic, Graves’ disease has well-established contributing factors. There is a strong genetic component, with increased prevalence...
19
Graves Disease II: Pathophysiology01:24

Graves Disease II: Pathophysiology

20
Graves’ disease is an autoimmune disorder characterized by the production of thyroid-stimulating immunoglobulins (TSI) that activate TSH receptors, leading to excessive synthesis and release of thyroid hormones (T3 and T4) and resulting in hyperthyroidism.Among all causes of hyperthyroidism, Graves’ disease is the most common and can happen at any age, though it is more frequent in women. It produces a hypermetabolic state with features such as weight loss, tachycardia, tremor,...
20
Hyperthyroidism I: Introduction01:25

Hyperthyroidism I: Introduction

28
Hyperthyroidism is a type of thyrotoxicosis characterized by the thyroid gland's overproduction of the thyroid hormones triiodothyronine (T3) and thyroxine (T4). This hormone excess increases the basal metabolic rate and enhances sensitivity to catecholamines.DiagnosisDiagnosis is based on clinical features and biochemical testing. It typically shows suppressed thyroid-stimulating hormone (TSH) levels below 0.4 mIU/L, with elevated free T3 and/or T4. Additional tests, including thyroid...
28
Hyperthyroidism II: Pathophysiology01:27

Hyperthyroidism II: Pathophysiology

26
Hyperthyroidism is a hypermetabolic state caused by elevated levels of thyroid hormones, triiodothyronine (T3) and thyroxine (T4). It results from dysregulation at the thyroid, pituitary, or immune system level and affects multiple organ systems.PathophysiologyThe most common cause of hyperthyroidism is Graves’ disease, an autoimmune disorder in which antibodies, specifically thyroid-stimulating antibodies (TSAb), a subtype of TSH receptor antibodies (TRAb), bind to and activate TSH...
26
Hypothyroidism II: Pathophysiology01:23

Hypothyroidism II: Pathophysiology

20
Hypothyroidism is a disorder characterized by insufficient production of thyroid hormones, which regulate metabolism, energy balance, and multiple organ systems.TypesHypothyroidism is classified based on the level of dysfunction. Primary hypothyroidism results from intrinsic thyroid gland dysfunction, causing reduced hormone production despite normal or increased stimulation. Secondary hypothyroidism arises from inadequate thyroid-stimulating hormone (TSH) secretion by the pituitary. Tertiary...
20
Synthesis and Regulation of Thyroid Hormones01:20

Synthesis and Regulation of Thyroid Hormones

7.1K
Low blood levels of the thyroid hormones — triiodothyronine (T3) and thyroxine (T4) — signal the hypothalamus to release the thyrotropin-releasing hormone (TRH). TRH then reaches the pituitary gland and stimulates the release of thyroid-stimulating hormone(TSH) into the bloodstream.
Upon reaching the thyroid gland, TSH stimulates the follicular cells' active uptake of iodide ions from the blood. The ions diffuse to the apical surface of the cells and are oxidized to iodine. The...
7.1K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

MAG-EX study protocol: magnetic stent versus stent on extraction string in patients after ureteroscopy-a multi-centre randomised controlled trial.

Translational andrology and urology·2026
Same author

Rivaroxaban Then Aspirin vs. Aspirin Alone after Total Hip or Knee Arthroplasty.

The New England journal of medicine·2026
Same author

Dissolving microneedle array size influences physical adjuvanticity for dose-sparing vaccination.

Biomaterials·2026
Same author

Management of Infected Wounds: A Wound Healing Foundation Consensus Statement.

Advances in wound care·2026
Same author

Solid microneedle booster as an enhanced microneedling platform for the treatment of androgenetic alopecia.

Drug delivery and translational research·2026
Same author

Advanced Practice Providers Versus Physicians in Emergency Department Abdominal Pain: Comparison of Length of Stay and Return Visits.

Academic emergency medicine : official journal of the Society for Academic Emergency Medicine·2026

Related Experiment Video

Updated: Apr 27, 2026

Minimal Invasive Resection of Large Retrosternal Thyroid Goiter
04:09

Minimal Invasive Resection of Large Retrosternal Thyroid Goiter

Published on: September 20, 2024

1.3K

Recurrent silent thyroiditis as a sequela of postpartum thyroiditis.

Preaw Hanseree1, Vincent Bryan Salvador1, Issac Sachmechi1

  • 1Department of Medicine, Icahn School of Medicine at Mount Sinai, Queens Hospital Center, Jamaica, NY 11432, USA.

Case Reports in Endocrinology
|July 3, 2014
PubMed
Summary

Recurrent silent thyroiditis can occur in women with a history of postpartum thyroiditis, even when not related to pregnancy. This case highlights the importance of considering silent thyroiditis in such patients.

More Related Videos

Generation of a Mouse Spontaneous Autoimmune Thyroiditis Model
04:39

Generation of a Mouse Spontaneous Autoimmune Thyroiditis Model

Published on: March 17, 2023

2.8K
Identification and Protection of the Recurrent Laryngeal Nerve during Transoral Robotic Thyroidectomy
05:25

Identification and Protection of the Recurrent Laryngeal Nerve during Transoral Robotic Thyroidectomy

Published on: October 24, 2025

943

Related Experiment Videos

Last Updated: Apr 27, 2026

Minimal Invasive Resection of Large Retrosternal Thyroid Goiter
04:09

Minimal Invasive Resection of Large Retrosternal Thyroid Goiter

Published on: September 20, 2024

1.3K
Generation of a Mouse Spontaneous Autoimmune Thyroiditis Model
04:39

Generation of a Mouse Spontaneous Autoimmune Thyroiditis Model

Published on: March 17, 2023

2.8K
Identification and Protection of the Recurrent Laryngeal Nerve during Transoral Robotic Thyroidectomy
05:25

Identification and Protection of the Recurrent Laryngeal Nerve during Transoral Robotic Thyroidectomy

Published on: October 24, 2025

943

Area of Science:

  • Endocrinology
  • Immunology

Background:

  • Thyroiditis is a group of thyroid inflammatory disorders.
  • Postpartum thyroiditis occurs within 12 months after delivery and is clinically similar to silent thyroiditis.
  • Recurrent postpartum thyroiditis is common, but recurrent silent thyroiditis is rare.

Purpose of the Study:

  • To report a case of recurrent silent thyroiditis in a patient with a history of postpartum thyroiditis.
  • To emphasize the potential association between postpartum thyroiditis and silent thyroiditis.
  • To alert physicians to the possibility of recurrent silent thyroiditis in women with prior postpartum thyroiditis.

Main Methods:

  • Case report presentation.
  • Clinical history review.
  • Diagnostic evaluation for thyroiditis.

Main Results:

  • A patient experienced recurrent episodes of thyroiditis unrelated to pregnancy after an initial episode of postpartum thyroiditis.
  • The findings suggest a potential link or co-occurrence between postpartum thyroiditis and silent thyroiditis.

Conclusions:

  • Postpartum thyroiditis and silent thyroiditis can occur sequentially in the same patient.
  • Physicians should consider the possibility of recurrent silent thyroiditis in women with a history of postpartum thyroiditis, even in the absence of pregnancy.