Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Diabetic neuropathy.

J D Ward

    British Medical Bulletin
    |January 1, 1989
    PubMed
    Summary
    This summary is machine-generated.

    Diabetic nerve damage stems from metabolic, microvascular, and aging factors. Improved nerve function measurement aids natural history and therapeutic studies, highlighting the need for standardized definitions.

    Related Concept Videos

    You might also read

    Related Articles

    Articles linked to this work by shared authors, journal, and citation graph.

    Sort by
    Same author

    The clinical impact of maternal COVID-19 on mothers, their infants, and placentas with an analysis of vertical transfer of maternal SARS-CoV-2-specific IgG antibodies.

    Placenta·2022
    Same author

    An engineered, orthogonal auxin analog/AtTIR1(F79G) pairing improves both specificity and efficacy of the auxin degradation system in Caenorhabditis elegans.

    Genetics·2021
    Same author

    Aerosolisation during tracheal intubation and extubation in an operating theatre setting.

    Anaesthesia·2020
    Same author

    Effects of methionine and lysine on fermentation in vitro and in vivo, nutrient flow to the intestine, and milk production.

    Journal of dairy science·2006
    Same author

    Sural nerve pathology in diabetic patients with minimal but progressive neuropathy.

    Diabetologia·2005
    Same author

    Effects of zinc and sodium monensin on ruminal degradation of lysine-HCl and liquid 2-hydroxy-4-methylthiobutanoic acid.

    Journal of dairy science·2004
    Same journal

    Human health in relation to visible, functional, and accessible green space: a systematic review and narrative synthesis of the 3 + 30 + 300 guideline.

    British medical bulletin·2026
    Same journal

    Muscle herniae in exercise-induced leg pain: diagnostic pitfalls and the 'repair paradox'.

    British medical bulletin·2026
    Same journal

    Scaffolds and platelet concentrates in bone regenerative medicine: applications, mechanisms, and future approaches.

    British medical bulletin·2026
    Same journal

    Back squat and deadlift fatiguing protocols elicit distinct countermovement jump profiles: phase-specific predictors and soreness responses.

    British medical bulletin·2026
    Same journal

    Enhancing bone healing through osteogenic medium components: biological roles, mechanisms, and clinical applications.

    British medical bulletin·2026
    Same journal

    Moral injury in healthcare workers: causes & interventions.

    British medical bulletin·2026
    See all related articles

    Area of Science:

    • Neurology
    • Endocrinology
    • Diabetology

    Background:

    • Diabetic neuropathy is a complex condition with multiple contributing factors.
    • Metabolic (myo-inositol), microvascular, glycosylation, and aging processes are implicated in nerve damage.
    • The diverse clinical presentations underscore the multifaceted nature of diabetic nerve complications.

    Purpose of the Study:

    • To review the contributing factors to diabetic nerve damage.
    • To highlight advancements in measuring peripheral and autonomic nerve function.
    • To emphasize the necessity for standardized clinical definitions and staging of diabetic neuropathy.

    Main Methods:

    • Review of existing literature on diabetic neuropathy.
    • Discussion of advancements in neurophysiological assessment techniques.

    Related Experiment Videos

  • Analysis of the implications of improved measurement for natural history and therapeutic research.

    • Main Results:

    • Multiple factors, including metabolic, microvascular, glycosylation, and aging, contribute to diabetic nerve damage.
    • Progress in measuring peripheral and autonomic nerve function is enabling better research.
    • Current understanding necessitates standardized approaches for clinical definition and staging.

    Conclusions:

    • Diabetic nerve damage is multifactorial, requiring a comprehensive understanding.
    • Advancements in functional measurements are crucial for future research and clinical trials.
    • Standardization of diagnostic criteria and staging is essential for consistent patient care and research.