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Postconditioning with Lactate-enriched Blood for Cardioprotection in ST-segment Elevation Myocardial Infarction
Published on: May 28, 2019
Post-cardiac injury syndrome after a simple coronary stenting
Yoshiaki Kawase1, Hideaki Ota, Munenori Okubo
1Department of Cardiovascular Medicine, Gifu Heart Center, 4-14-4 Yabutaminami, Gifu City, Gifu, 500-8384, Japan, ykawase@heart-center.or.jp.
Insights
Post-coronary angioplasty, a patient developed chest pain and fluid buildup, later diagnosed as cardiac tamponade. Treatment with pericardiocentesis and corticosteroids resolved the condition, suggesting post-cardiac injury syndrome.
Area of Science:
- Cardiology
- Internal Medicine
Background:
- Coronary angioplasty is a common procedure for treating coronary artery disease.
- Post-procedural complications, though rare, require prompt diagnosis and management.
Observation:
- An 80-year-old male presented with chest pain and ST elevation post-coronary angioplasty.
- Initial symptoms included pulmonary congestion and pleural effusion, managed with diuretics.
- Recurrent symptoms led to the diagnosis of cardiac tamponade.
Findings:
- Echocardiography initially showed no significant pericardial effusion but later confirmed cardiac tamponade.
- Pericardiocentesis and pleurocentesis improved oxygenation.
- Intravenous hydrocortisone followed by oral prednisone led to drastic clinical improvement.
Implications:
- This case highlights the importance of considering post-cardiac injury syndrome in patients with delayed post-procedural complications.
- Prompt diagnosis and multi-modal treatment, including pericardiocentesis and corticosteroids, are crucial for favorable outcomes.
- Further research into the early detection and management of this syndrome is warranted.
Abstract:
An 80-year-old male patient underwent a coronary angioplasty without signs of complications. The day after the procedure, the patient complained of chest pain. The electrocardiogram showed a widespread ST segment elevation. A chest X-ray revealed pulmonary congestion with pleural effusion. There was no significant pericardial effusion detected with an echocardiogram. An administration of diuretics was initiated. After he showed an improvement of symptoms, the administration of diuretics was tapered. However, a deterioration of the oxygenation level was re-observed. The echocardiogram confirmed a cardiac tamponade at this time. The oxygenation level recovered after pericardiocentesis and pleurocentesis. Post-cardiac injury syndrome was suspected to be the cause of this clinical course, and the patient was given an intravenous administration of hydrocortisone followed by an oral administration of prednisone. All clinical parameters started to improve drastically.
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