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Insulin-like growth factor-I in the parenterally fed low birth weight infant

J R Saini1, J B Morgan, J D Teale

  • 1Department of Child Health, Charing Cross and Westminster Medical School, London, UK.

Insights

Insulin-like growth factor-1 (IGF-I) levels often decreased with age in very low birth weight infants receiving parenteral nutrition. Low IGF-I may indicate poor nutritional status in these vulnerable preterm infants.

Area of Science:

  • Neonatalogy
  • Pediatric Endocrinology
  • Nutritional Science

Background:

  • Very low birth weight (VLBW) infants (<1200g, <30 weeks gestation) often experience growth faltering.
  • Parenteral nutrition (PN) is crucial for VLBW infants but its impact on growth factors requires further study.
  • Insulin-like growth factor-1 (IGF-I) is a key regulator of growth and development.

Purpose of the Study:

  • To investigate the longitudinal changes in insulin-like growth factor-1 (IGF-I) in VLBW infants receiving predominantly parenteral nutrition.
  • To explore potential relationships between IGF-I levels, nutritional intake, and nitrogen retention in this population.

Main Methods:

  • Longitudinal study of 9 VLBW infants (<1200g, <30 weeks gestation).
  • Infants received ≥85% of nutrient intake via parenteral route.
  • IGF-I levels were measured over time and correlated with nitrogen intake, energy intake, and nitrogen retention.

Main Results:

  • No significant relationship was observed between IGF-I levels and nitrogen intake, energy intake, or nitrogen retention.
  • In 6 out of 9 infants, IGF-I levels showed a decrease with increasing postnatal age.
  • IGF-I levels varied considerably among the studied infants.

Conclusions:

  • Decreasing IGF-I levels with postnatal age in VLBW infants on PN may not be directly linked to specific nutritional parameters studied.
  • Low IGF-I levels in sick preterm infants could be a marker of their overall poor nutritional status and compromised growth rate.
  • Further research is needed to elucidate the complex factors influencing IGF-I regulation in VLBW infants.

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