Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Transient Ischemic Attack l: Introduction01:26

Transient Ischemic Attack l: Introduction

13
A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
13
Stroke: Introduction and Types01:29

Stroke: Introduction and Types

39
A stroke is an acute neurological event caused by the sudden disruption of cerebral blood flow, leading to rapid loss of neuronal function. Neurons depend on continuous oxygen and glucose supply, so even brief interruptions can cause irreversible injury within minutes. Strokes are classified into ischemic and hemorrhagic types.Ischemic StrokeIschemic strokes are most common and occur due to arterial occlusion, depriving brain tissue of oxygen and nutrients. This leads to energy failure, ionic...
39
Ischemic Stroke l: Introduction01:15

Ischemic Stroke l: Introduction

29
Ischemic stroke is an acute cerebrovascular condition in which blood flow to a brain region is suddenly interrupted, leading to tissue infarction. Neurons depend on continuous oxygen and glucose supply, so even brief reductions in perfusion cause energy failure, ionic imbalance, and irreversible injury. Ischemic strokes are classified into thrombotic and embolic types based on their underlying mechanisms.Thrombotic MechanismsThrombotic stroke develops when a clot forms within a cerebral artery.
29
Ischemic Stroke ll: Pathophysiology01:15

Ischemic Stroke ll: Pathophysiology

42
An ischemic stroke occurs when a cerebral blood vessel becomes obstructed, most often by a thrombus or embolus, interrupting the delivery of oxygen and glucose to brain tissue. Because neurons rely on continuous aerobic metabolism, energy failure begins within minutes of reduced perfusion. The region receiving the least blood flow becomes the infarct core, an area of irreversible cellular death. Surrounding this core lies the penumbra, a zone of hypoperfused but still viable tissue that is...
42
Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

14
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
14
Epilepsy ll: Types01:22

Epilepsy ll: Types

27
Recurrent seizures, stemming from abnormal electrical activity in the brain, are the defining characteristic of epilepsy, a chronic neurological condition. Because seizure features vary greatly, epilepsy is classified using two systems: by seizure type and by epilepsy syndromes. These classifications enable clinicians to describe seizure patterns and select suitable treatment strategies.I. Classification by Seizure Type1. Focal EpilepsyFocal epilepsy begins in one hemisphere of the brain.
27

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Comparison of infliximab with adalimumab in biologic-naïve patients with Crohn's disease: a single-center 13-year experience.

European review for medical and pharmacological sciences·2023
Same author

Association of obesity with recurrent stroke and cardiovascular events.

Revue neurologique·2020
Same author

Limb shaking transient ischemic attacks: A follow-up of 28 patients.

Revue neurologique·2020
Same author

Opacification of nondilated bile ducts through the gallbladder as an aid to percutaneous transhepatic biliary drainage.

Diagnostic and interventional imaging·2017
Same author

A comparative study of art therapy in cancer patients receiving chemotherapy and improvement in quality of life by watercolor painting.

Complementary therapies in medicine·2017
Same author

Increased Serum Soluble CD23 and Soluble IL-2R Levels in Haematologic Malignancies.

Turkish journal of haematology : official journal of Turkish Society of Haematology·2016

Related Experiment Video

Updated: Apr 26, 2026

Brain Infarct Segmentation and Registration on MRI or CT for Lesion-symptom Mapping
10:25

Brain Infarct Segmentation and Registration on MRI or CT for Lesion-symptom Mapping

Published on: September 25, 2019

48.8K

Multiple variant type thalamic infarcts: pure and combined types.

E Kumral1, E E Deveci, A Y Çolak

  • 1Stroke Unit, Neurology Department, School of Medicine, Ege University, İzmir, Turkey.

Acta Neurologica Scandinavica
|August 12, 2014
PubMed
Summary

This study identifies distinct clinical, etiological, and radiological features of variant thalamic infarcts, highlighting their unique patterns and potential causes like arterial supply variations or embolism.

Keywords:
MRIlarge-artery diseasesmall-artery diseasethalamic infarct

More Related Videos

Assessing Cortical Cerebral Microinfarcts on High Resolution MR Images
08:39

Assessing Cortical Cerebral Microinfarcts on High Resolution MR Images

Published on: November 20, 2015

12.8K
Combined Near-infrared Fluorescent Imaging and Micro-computed Tomography for Directly Visualizing Cerebral Thromboemboli
13:10

Combined Near-infrared Fluorescent Imaging and Micro-computed Tomography for Directly Visualizing Cerebral Thromboemboli

Published on: September 25, 2016

9.4K

Related Experiment Videos

Last Updated: Apr 26, 2026

Brain Infarct Segmentation and Registration on MRI or CT for Lesion-symptom Mapping
10:25

Brain Infarct Segmentation and Registration on MRI or CT for Lesion-symptom Mapping

Published on: September 25, 2019

48.8K
Assessing Cortical Cerebral Microinfarcts on High Resolution MR Images
08:39

Assessing Cortical Cerebral Microinfarcts on High Resolution MR Images

Published on: November 20, 2015

12.8K
Combined Near-infrared Fluorescent Imaging and Micro-computed Tomography for Directly Visualizing Cerebral Thromboemboli
13:10

Combined Near-infrared Fluorescent Imaging and Micro-computed Tomography for Directly Visualizing Cerebral Thromboemboli

Published on: September 25, 2016

9.4K

Area of Science:

  • Neurology
  • Neuroimaging
  • Stroke Medicine

Background:

  • Thalamic infarcts exhibit variant topographic patterns classified into anteromedian, central, and posterolateral territories.
  • Understanding these distinct patterns is crucial for accurate diagnosis and management.

Purpose of the Study:

  • To determine the clinical, etiological, and radiological features of multiple variant thalamic infarcts.
  • To delineate specific patterns of thalamic infarction outside classical territories.

Main Methods:

  • Retrospective review of 8400 first-ever stroke patients from the Ege Stroke Registry (2000-2013).
  • Selection of 21 patients with acute multiple thalamic infarcts outside classical territories, confirmed by MRI.
  • Analysis of clinical, etiological, and radiological data.

Main Results:

  • 21 patients (26% of multiple thalamic infarcts) presented with variant infarct distributions.
  • Unilateral infarcts (7 patients) showed decreased vigilance, cognitive impairment, and aphasia; cardioembolism was the main cause (43%).
  • Bilateral infarcts (5 patients) presented with consciousness disturbances and sensory-motor deficits; cardioembolism was the main cause (60%).
  • Combined infarcts (9 patients) showed hemihypesthesia and hemiataxia; cardioembolism (56%) and vertebrobasilar disease (33%) were key mechanisms.

Conclusions:

  • Multiple variant thalamic infarcts have distinct manifestations and etiologies.
  • These infarcts may result from variations in thalamic arterial supply or embolism sources.