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Development of low postural tone compensatory patterns - predicted dysfunction patterns in upper part of the body
Anna Gogola1, Edward Saulicz, Michał Kuszewski
1Katedra Kinezyterapii i Metod Specjalnych Fizjoterapii, Akademia Wychowania Fizycznego, ul. Mikołowska 72b, 40-065 Katowice, tel. 603-112-533,
Reduced deep muscle tone, even within normal ranges, can lead to specific postural faults through active or passive compensation mechanisms. These compensatory strategies impact joint mobility and spinal alignment.
Area of Science:
- Kinesiology
- Human Physiology
- Biomechanics
Background:
- Lower postural tone is not always indicative of central nervous system damage.
- A decrease in deep stabilizing muscle tone can occur within normal ranges.
- This subtle tone reduction can initiate compensatory postural and motor patterns.
Purpose of the Study:
- To investigate the compensatory mechanisms triggered by reduced deep muscle tone.
- To differentiate between active and passive compensation strategies.
- To identify the resulting postural faults associated with these compensation types.
Main Methods:
- Observational analysis of postural and motor patterns.
- Identification of active compensation features (superficial muscle overuse, joint restriction).
- Identification of passive compensation features (skeletal, ligamentous, meniscal reliance, hypermobility).
Main Results:
- Active compensation leads to head translation, elevated shoulder girdle, and increased thoracic kyphosis (spastoidal type).
- Passive compensation results in decreased thoracic kyphosis, sunken chest, and increased joint range of motion (atetoidal type).
- Both compensation types stem from reduced tone in stabilizing muscles, leading to distinct postural deviations.
Conclusions:
- Reduced tone in deep stabilizing muscles initiates a cascade of compensatory mechanisms.
- These mechanisms, whether active or passive, result in specific and identifiable postural faults.
- Understanding these compensatory patterns is crucial for addressing postural deviations not linked to neurological damage.
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