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High-density lipoprotein subfractions and carotid plaque: the Northern Manhattan Study

Eduard Tiozzo1, Hannah Gardener2, Barry I Hudson3

  • 1Department of Psychiatry and Behavioral Sciences, Miller School of Medicine, University of Miami, Miami, FL, USA.

Atherosclerosis
|September 21, 2014
PubMed

Insights

High-density lipoprotein cholesterol (HDL-C) subfractions HDL3-C and HDL2-C show distinct associations with carotid plaque characteristics. Lower HDL3-C relates to reduced plaque area, while higher HDL2-C correlates with increased plaque thickness, impacting vascular disease risk.

Area of Science:

  • Cardiovascular Medicine
  • Lipid Metabolism
  • Vascular Biology

Background:

  • High-density lipoprotein cholesterol (HDL-C) plays a crucial role in reverse cholesterol transport.
  • HDL-C exists in distinct subfractions, HDL2-C and HDL3-C, with potentially differential atherogenic roles.
  • Understanding these subfractions' associations with subclinical atherosclerosis is vital for cardiovascular risk assessment.

Purpose of the Study:

  • To investigate the relationship between HDL2-C and HDL3-C levels and carotid plaque characteristics.
  • To analyze associations with carotid plaque area and thickness in a population-based cohort.
  • To explore potential differential impacts of HDL subfractions on vascular disease risk.

Main Methods:

  • Cross-sectional analysis of 988 stroke-free participants (mean age 66 ± 8 years).
  • HDL subfractions (HDL2-C, HDL3-C) measured using a precipitation method.
  • Carotid plaque area and thickness assessed via high-resolution 2D ultrasound; quantile regression used for analysis.

Main Results:

  • Carotid plaque was present in 56% of participants.
  • Inverse association found between HDL3-C and plaque area (p=0.001 at 75th percentile).
  • Positive association observed between HDL2-C and plaque thickness (p=0.003 at 90th percentile).
  • Total HDL-C associated with plaque area only in Hispanic participants (p=0.01).

Conclusions:

  • HDL3-C is inversely associated with carotid plaque area.
  • HDL2-C is positively associated with carotid plaque thickness.
  • HDL subfractions may have distinct roles in atherogenesis, suggesting potential for targeted HDL-based therapies.
Abstract

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