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Related Concept Videos

Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

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A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
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Hemorrhagic Stroke l: Introduction01:17

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A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
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Related Experiment Video

Updated: Apr 22, 2026

Application of Laparoscopic Partial Splenectomy with Total Blood Flow Occlusion in Benign Splenic Lesions
02:09

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Published on: December 20, 2024

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Giant splenic hematoma can be a hidden condition.

Kristian Kiim Jensen, Stine Hangaard

    Polski Przeglad Chirurgiczny
    |October 9, 2014
    PubMed
    Summary

    A healthy male experienced morning dyspnoea and abdominal pain due to a large splenic hematoma, diagnosed four months after abdominal trauma. Conservative treatment was initiated for this significant spleen injury.

    Area of Science:

    • Traumatology
    • Abdominal Imaging
    • Hematology

    Background:

    • A 28-year-old male with no prior health issues presented with chronic abdominal discomfort and morning dyspnoea.
    • Symptoms began months after sustaining blunt abdominal trauma to the left side.
    • Initial presentation suggested a potential link between trauma and subsequent symptoms.

    Observation:

    • Abdominal ultrasonography identified a splenic hematoma.
    • Computed tomography (CT) confirmed an 18-centimeter splenic hematoma.
    • Laboratory tests revealed marginal anemia and elevated C-reactive protein.

    Findings:

    • The patient was diagnosed with a large splenic hematoma secondary to blunt abdominal trauma.
    • Despite significant size, vital signs remained stable, and other bloodwork was largely unremarkable.

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  • Conservative management was chosen for the splenic hematoma.

    • Implications:

    • This case highlights the potential for delayed presentation of splenic hematomas after trauma.
    • It underscores the importance of advanced imaging like CT in diagnosing significant internal injuries.
    • Conservative management can be a viable option for splenic hematomas in hemodynamically stable patients.