Orexin-A stimulates 3β-hydroxysteroid dehydrogenase expression and cortisol production in H295R human adrenocortical

Xiaocen Chang1, Yuyan Zhao1, Shujing Ju1

  • 1Departments of Endocrinology and Orthopedic Surgery, The First Affiliated Hospital, China Medical University, Shenyang, Liaoning 110001, P.R. China.

Insights

Orexin-A influences human adrenocortical cells by increasing proliferation and cortisol production via the OX1 receptor and AKT pathway. These orexin-A effects on cell survival and function are mediated through AKT signaling.

Area of Science:

  • Endocrinology
  • Cell Biology
  • Molecular Signaling

Background:

  • Orexin-A is a peptide regulating appetite, sleep, and metabolic functions.
  • It acts through orexin receptor type 1 (OX1 receptor) and orexin receptor type 2 (OX2 receptor).
  • The role of orexin-A in adrenocortical cell function is not fully understood.

Purpose of the Study:

  • To investigate the expression of OX1 receptor in human adrenocortical cells.
  • To determine the effects of orexin-A on adrenocortical cell proliferation, cortisol production, and 3β-HSD expression.
  • To elucidate the involvement of the AKT signaling pathway in mediating orexin-A's actions on adrenocortical cells.

Main Methods:

  • Human adrenocortical NCI-H295R cells were treated with varying concentrations of orexin-A.
  • OX1 receptor mRNA and protein expression were analyzed.
  • Cell proliferation, cortisol content, 3β-HSD expression, and AKT pathway activity were assessed following treatment with orexin-A and specific antagonists (SB334867 for OX1 receptor, PF-04691502 for AKT).

Main Results:

  • Orexin-A increased OX1 receptor mRNA and protein expression in a concentration-dependent manner.
  • Orexin-A enhanced NCI-H295R cell proliferation, cortisol production, and 3β-HSD expression.
  • These effects were partially inhibited by OX1 receptor and AKT antagonists.
  • Orexin-A activated the AKT signaling pathway, evidenced by increased AKT phosphorylation, an effect blocked by antagonists.

Conclusions:

  • Orexin-A upregulates OX1 receptor expression in human adrenocortical cells.
  • Orexin-A promotes adrenocortical cell survival and function, including cortisol production.
  • The effects of orexin-A on adrenocortical cells are mediated via the OX1 receptor and the AKT signaling pathway.

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