Modes of induced cardiac arrest: hyperkalemia and hypocalcemia--literature review
Marcos Aurélio Barboza de Oliveira1, Antônio Carlos Brandi2, Carlos Alberto dos Santos2
1Faculdade de Medicina de São José do Rio Preto, São José do Rio Preto, SP, Brasil.
Abstract:
The entry of sodium and calcium play a key effect on myocyte subjected to cardiac arrest by hyperkalemia. They cause cell swelling, acidosis, consumption of adenosine triphosphate and trigger programmed cell death. Cardiac arrest caused by hypocalcemia maintains intracellular adenosine triphosphate levels, improves diastolic performance and reduces oxygen consumption, which can be translated into better protection to myocyte injury induced by cardiac arrest.
