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Characterization of the bacterial cell associated calmodulin-sensitive adenylate cyclase from Bordetella pertussis

H R Masure1, D R Storm

  • 1Department of Pharmacology, School of Medicine, University of Washington, Seattle 98195.

Biochemistry
|January 24, 1989
PubMed

Insights

Bordetella pertussis adenylate cyclase is released from bacteria and invades cells, increasing cAMP. The enzyme

Area of Science:

  • Microbiology
  • Biochemistry
  • Molecular Biology

Background:

  • Bordetella pertussis secretes adenylate cyclase, an enzyme that elevates intracellular cAMP levels in host cells.
  • This calmodulin-sensitive adenylate cyclase exists in both cell-associated and released forms.

Purpose of the Study:

  • To investigate the localization and forms of Bordetella pertussis adenylate cyclase.
  • To understand the relationship between the different molecular weight forms of the enzyme.

Main Methods:

  • SDS-polyacrylamide gel electrophoresis to resolve enzyme forms.
  • Antibody cross-reactivity and immunoprecipitation assays.
  • Enzyme activity assays measuring cAMP levels.

Main Results:

  • Cell-associated adenylate cyclase (28%) is mostly external, sensitive to trypsin.
  • SDS treatment releases a large form (215,000 Da) from cells; culture supernatant contains a smaller catalytic subunit (45,000 Da).
  • Proteolytic degradation likely produces the smaller form; antibodies recognize both forms.

Conclusions:

  • Bordetella pertussis adenylate cyclase has a large cell-associated form and a smaller catalytic subunit released extracellularly.
  • The enzyme's catalytic domain is on the bacterial surface, and it can be processed into smaller forms.
  • These forms contribute to the enzyme's ability to increase intracellular cAMP.

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