Related Experiment Video

Updated: Apr 20, 2026

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model
09:14

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model

Published on: June 18, 2021

3.0K

Subarachnoid hemorrhage due to vascular causes and gnathostomiasis: clinical features and laboratory findings

    The Southeast Asian Journal of Tropical Medicine and Public Health
    |November 25, 2014
    PubMed

    Subarachnoid hemorrhage (SAH) is a serious neurological condition, commonly of vascular etiology. Gnathostomiasis is a common parasitic disease in Thailand and may also cause SAH. The purpose of this study was to find clinical differences between SAH due to these two causes. This was a retrospective study and collected data from medical charts of patients diagnosed with SAH at Srinagarind Hospital, Khon Kaen, during 2009 and 2011. SAH due to vascular causes was diagnosed by cerebral angiogram, while cerebral gnathostomiasis, in which cerebral angiograms were negative, was diagnosed immunologically. Differences in clinical features between the two groups were compared using de- scriptive statistics. Eighteen patients had SAH due to vascular causes and ten had gnathostomiasis. Most parameters were similar between the two groups. However, the cerebrospinal fluid glucose/plasma glucose ratio in the gnathostomiasis group was significantly higher than in the vascular group (80% vs. 16.67%, respectively). In conclusion, cerebrospinal fluid glucose/plasma glucose ratio was significantly higher in SAH patients caused by gnathostomiasis than vascular group and may provide a diagnostic tool for distinguishing between these two etiologies.

    More Related Videos

    Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage10:34

    Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage

    Published on: August 30, 2020

    11.8K
    A Low Mortality Rat Model to Assess Delayed Cerebral Vasospasm After Experimental Subarachnoid Hemorrhage07:03

    A Low Mortality Rat Model to Assess Delayed Cerebral Vasospasm After Experimental Subarachnoid Hemorrhage

    Published on: January 17, 2013

    14.9K

    Related Concept Videos

    Hemorrhagic Stroke l: Introduction01:17

    Hemorrhagic Stroke l: Introduction

    A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
    4
    Hemorrhagic Stroke ll: Pathophysiology01:29

    Hemorrhagic Stroke ll: Pathophysiology

    A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
    6
    Bacterial Meningitis II: Pathophysiology01:26

    Bacterial Meningitis II: Pathophysiology

    Bacterial meningitis typically begins when pathogens such as Neisseria meningitidis and Streptococcus pneumoniae colonize the nasopharynx and invade the bloodstream. This process is facilitated by bacterial virulence factors, such as polysaccharide capsules, which resist phagocytosis and complement-mediated killing. Less commonly, bacteria reach the central nervous system via contiguous spread from infections like otitis media or sinusitis, through congenital or acquired dural defects, or...
    2
    Aneurysm II: Clinical Manifestations and Diagnostic Studies01:21

    Aneurysm II: Clinical Manifestations and Diagnostic Studies

    Thoracic, aortic arch and abdominal aneurysms are significant vascular conditions that can present with various clinical manifestations and lead to serious complications. Understanding these manifestations and the appropriate diagnostic studies is essential for effective management and treatment.Thoracic Aortic AneurysmsThoracic aortic aneurysms often remain asymptomatic until they reach a size that impinges on adjacent structures. They typically cause deep, diffuse chest pain that radiates to...
    564
    Bacterial Meningitis I: Introduction01:22

    Bacterial Meningitis I: Introduction

    Bacterial meningitis is a severe, life-threatening inflammation of the meninges, particularly the pia mater and arachnoid mater, affecting the subarachnoid space, ventricles, and cerebrospinal fluid (CSF). If untreated, it can lead to significant neurological complications or death.Causative AgentsCommon pathogens vary with age and immune status. In adults, major organisms include Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. Streptococcus agalactiae (group B...
    4
    Venous Thrombosis II: Clinical Manifestations and Diagnostic Studies01:20

    Venous Thrombosis II: Clinical Manifestations and Diagnostic Studies

    The key difference between Superficial Vein Thrombosis (SVT) and Deep Vein Thrombosis (DVT) lies in their location and severity.Clinical ManifestationsSVT typically presents with localized pain, tenderness, and redness along the course of a superficial vein, often accompanied by a palpable, cord-like structure under the skin. This condition is usually less dangerous than DVT but can be uncomfortable and may lead to complications such as cellulitis or, rarely, a clot extension into the deep...
    451