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The N- and C-terminal autolytic fragments of CAPN3/p94/calpain-3 restore proteolytic activity by intermolecular

Yasuko Ono1, Mayumi Shindo2, Naoko Doi3

  • 1Department of Advanced Science for Biomolecules, Calpain Project, and Cellular and Molecular Medicine and Sarver Molecular Cardiovascular Research Program, University of Arizona, Tucson, AZ 85724 ono-ys@igakuken.or.jp.

Proceedings of the National Academy of Sciences of the United States of America
|December 17, 2014
PubMed

Keywords:
calpaincomplementationmuscular dystrophyproteaseskeletal muscle

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