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Related Concept Videos

Acute Pyelonephritis II: Diagnostic Studies and Management01:28

Acute Pyelonephritis II: Diagnostic Studies and Management

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Introduction:For diagnosing acute pyelonephritis, a comprehensive patient history is collected to identify symptoms such as dysuria, frequent or urgent urination, flank pain, or costovertebral angle (CVA) tenderness that may suggest a kidney infection.Physical ExaminationDuring the physical examination, CVA tenderness is assessed. This involves gentle percussion over the costovertebral angle, where tenderness often indicates a kidney infection.Diagnostic TestsUrinalysis: Used to identify white...
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Staphylococcal Skin Infections01:29

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Staphylococcus aureus is a Gram-positive coccus that resides harmlessly on the skin and mucous membranes of healthy individuals. When the skin barrier is breached, it can shift from a commensal to an opportunistic pathogen. This transition is facilitated by surface adhesins, such as clumping factor B and S. aureus surface protein G (SasG), which bind to structural proteins, including loricrin and cytokeratin, in the damaged epidermis. Protein A, another key factor, binds the Fc region of...
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Bacterial Meningitis I: Introduction01:22

Bacterial Meningitis I: Introduction

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Bacterial meningitis is a severe, life-threatening inflammation of the meninges, particularly the pia mater and arachnoid mater, affecting the subarachnoid space, ventricles, and cerebrospinal fluid (CSF). If untreated, it can lead to significant neurological complications or death.Causative AgentsCommon pathogens vary with age and immune status. In adults, major organisms include Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. Streptococcus agalactiae (group B...
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Brain Abscess l: Introduction01:26

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A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial...
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Bacterial Meningitis II: Pathophysiology01:26

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Bacterial meningitis typically begins when pathogens such as Neisseria meningitidis and Streptococcus pneumoniae colonize the nasopharynx and invade the bloodstream. This process is facilitated by bacterial virulence factors, such as polysaccharide capsules, which resist phagocytosis and complement-mediated killing. Less commonly, bacteria reach the central nervous system via contiguous spread from infections like otitis media or sinusitis, through congenital or acquired dural defects, or...
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Acute Pyelonephritis I: Introduction01:27

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Pyelonephritis is a bacterial infection that primarily affects the renal parenchyma and collecting system, including the renal pelvis, tubules, and interstitial tissue of one or both kidneys. It can be classified as either acute—a sudden, severe infection—or chronic, which refers to long-term or recurrent kidney infections.The primary cause of acute pyelonephritis (APN) is bacterial infection, with Escherichia coli accounting for approximately 70-80% of cases. Other bacteria, such...
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Updated: Apr 18, 2026

A Murine Model of Group B Streptococcus Vaginal Colonization
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A Murine Model of Group B Streptococcus Vaginal Colonization

Published on: November 16, 2016

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Group G Streptococcus bacteremia in recurrent cellulitis.

Nicola di Meo1, Giuseppe Stinco, Nicoletta Gubertini

  • 1Nicola di Meo, MD, Dermatology Department, University of Trieste , Ospedale Maggiore di Trieste, IV piano Palazzina Infettiv,i Piazza Ospedale 1, 34151 Trieste, Italy; nickdimeo@libero.it.

Acta Dermatovenerologica Croatica : ADC
|January 13, 2015
PubMed
Summary
This summary is machine-generated.

Group G Streptococcus infections are increasingly common, sometimes causing severe toxic effects. Prompt antibiotic treatment and long-term penicillin prophylaxis effectively manage recurrent group G Streptococcus cellulitis.

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A Murine Model of Group B Streptococcus Vaginal Colonization
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Published on: November 16, 2016

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Area of Science:

  • Infectious Diseases
  • Bacteriology
  • Clinical Medicine

Background:

  • Group G Streptococcus is an emerging cause of human infections.
  • Immunosuppression, malignancy, and diabetes are predisposing host factors.
  • Toxic involvement and sequelae are now recognized in Group G and C Streptococcus infections.

Observation:

  • A case of recurrent cellulitis with group G Streptococcus bacteremia and toxic involvement is presented.
  • Blood cultures were negative for beta-hemolytic Streptococci during recurrences, except the final one.
  • The patient experienced recurrent cellulitis despite initial negative blood cultures.

Findings:

  • Antibiotic therapy guided by antibiogram rapidly resolved the acute infection.
  • Intramuscular benzathine penicillin (1.2 million units every 15 days for one year) prevented cellulitis recurrence.
  • This regimen proved effective in managing recurrent group G Streptococcus infections.

Implications:

  • Group G Streptococcus infections require vigilant diagnosis and management.
  • Understanding predisposing factors is crucial for preventing severe outcomes.
  • Effective treatment strategies, including long-term prophylaxis, can prevent recurrence and sequelae.