Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Dysrhythmias IV: Characteristics of Bradyarrhythmias01:18

Dysrhythmias IV: Characteristics of Bradyarrhythmias

845
Bradyarrhythmias are cardiac rhythm disorders characterized by a slower-than-normal heart rate, typically defined as fewer than 60 beats per minute. Some of which are discussed here:Sinus BradycardiaSinus bradycardia presents a heart rate lower than 60 beats per minute, with a regular rhythm originating from the SA node. The ECG typically shows normal P waves preceding each QRS complex, a normal PR interval (0.12 to 0.20 seconds), and a normal QRS duration (0.06 to 0.10 seconds).First-Degree AV...
845
Disturbances in Heart Rhythm01:29

Disturbances in Heart Rhythm

5.4K
Arrhythmia or dysrhythmia refers to an abnormal heart rhythm caused by a defect in the heart's conduction system. It can cause the heart to beat irregularly, too quickly, or too slowly, leading to symptoms like chest pain, shortness of breath, and fainting. Factors such as stress, caffeine, alcohol, nicotine, cocaine, certain drugs, congenital defects, diseases, and electrolyte abnormalities can trigger arrhythmias.
Arrhythmias are categorized by their speed, rhythm, and origin. A slow heart...
5.4K
ECG Interpretation of Arrhythmias II: Atrial, Junctional and Ventricular Arrhythmias01:25

ECG Interpretation of Arrhythmias II: Atrial, Junctional and Ventricular Arrhythmias

1.1K
Arrhythmia is a condition characterized by an irregular heart rhythm, with ECG changes that differ based on its origin and nature. The types of arrhythmias discussed below include atrial, junctional, and ventricular arrhythmias.Atrial ArrhythmiasPremature Atrial Complexes (PACs): PACs are early atrial beats caused by stress, caffeine, alcohol, electrolyte imbalances, hypoxia, hyperthyroidism, or certain medications (e.g., bronchodilators and decongestants). The ECG shows early P waves with an...
1.1K
Dysrhythmias II: Classification of Tachyarrhythmias01:28

Dysrhythmias II: Classification of Tachyarrhythmias

798
Tachyarrhythmias are a type of dysrhythmia where the heart rate exceeds 100 beats per minute. Here are some common types of tachyarrhythmias:Sinus TachycardiaSinus tachycardia originates from increased impulses from the sinus node, leading to an elevated heart rate. It is often triggered by stress, fever, or exercise.Patients may experience palpitations, a sensation of a racing heart, dizziness, and chest discomfort.Causes and Risk Factors: Common causes include physical exertion, emotional...
798
Antiarrhythmic Drugs: Class II Agents as β-Adrenergic Blockers01:24

Antiarrhythmic Drugs: Class II Agents as β-Adrenergic Blockers

2.4K
Adrenergic stimulation generally impacts cardiac rate and rhythm. Specifically, stimulation of the β-adrenoceptors triggers an increase in intracellular calcium ion influx and pacemaker currents, which may cause arrhythmias. Catecholamines like adrenaline also demonstrate β2-adrenoceptor-mediated hypokalemia, impacting cardiac action potential and disrupting the normal cardiac rhythm. Class II antiarrhythmic drugs are β-adrenoceptor antagonists or β-blockers, which...
2.4K
Antiarrhythmic Drugs: Class IV Agents as Calcium Channel Blockers01:20

Antiarrhythmic Drugs: Class IV Agents as Calcium Channel Blockers

2.8K
Class IV antiarrhythmic drugs, such as verapamil and diltiazem, block calcium channels. They primarily affect the heart, slowing the conduction in calcium-dependent tissues like the SA and AV nodes. These drugs manage reentrant supraventricular tachycardia (SVT) and reduce ventricular rate in atrial flutter/fibrillation.
Verapamil, a calcium channel blocker, inhibits calcium movement across myocardial cell membranes and vascular smooth muscle. This results in the dilation of coronary and...
2.8K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Dislodged Right Ventricular Pacemaker Lead Detected Incidentally Two Years Post-implantation via Echocardiography: A Case Report.

Cureus·2025
Same author

Role of Medication Reconciliation in Managing Bradycardia: A Rare Cause of Medication-Induced Sinus Bradycardia.

Cureus·2025
Same author

Impact of Obesity in Hospitalized Patients Undergoing Catheter Ablation for Atrial Fibrillation.

Pacing and clinical electrophysiology : PACE·2025
Same author

Dual-morphology Bundle Branch Re-entrant Ventricular Tachycardia in Non-dilated Cardiomyopathy.

The Journal of innovations in cardiac rhythm management·2025
Same author

Metformin: Its salutary effects beyond diabetes mellitus.

Journal of investigative medicine : the official publication of the American Federation for Clinical Research·2025
Same author

Oral theophylline corrects sinus node dysfunction in acute on chronic lithium toxicity: case report and systematic review of lithium-induced Sinus node dysfunction.

Frontiers in cardiovascular medicine·2024

Related Experiment Video

Updated: Apr 18, 2026

Non-fluoroscopic Catheter Tracking for Fluoroscopy Reduction in Interventional Electrophysiology
10:46

Non-fluoroscopic Catheter Tracking for Fluoroscopy Reduction in Interventional Electrophysiology

Published on: May 26, 2015

14.0K

Recurrent A V block following ablation for AVNRT.

Fnu Shailesh1, Asif Sewani1, Hakan Paydak1

  • 1Department of Cardiovascular Medicine, University of Arkansas for Medical Sciences, 4301, W Markham St, # 634, Little Rock, AR 72205-7199, USA.

Indian Heart Journal
|January 31, 2015
PubMed
Summary

Radiofrequency ablation for atrio-ventricular nodal re-entrant tachycardia (AVNRT) can rarely cause delayed complete heart block. This transient complication, occurring weeks after the procedure, requires close patient monitoring.

Keywords:
Atrio-ventricular nodal re-entrant tachycardia (AVNRT)Electrocardiogram (ECG)Heart blockRadiofrequency ablation (RFA)Slow pathway (SP)

More Related Videos

Robotic Ablation of Atrial Fibrillation
11:21

Robotic Ablation of Atrial Fibrillation

Published on: May 29, 2015

20.4K
Remote Magnetic Navigation for Accurate, Real-time Catheter Positioning and Ablation in Cardiac Electrophysiology Procedures
09:13

Remote Magnetic Navigation for Accurate, Real-time Catheter Positioning and Ablation in Cardiac Electrophysiology Procedures

Published on: April 21, 2013

28.7K

Related Experiment Videos

Last Updated: Apr 18, 2026

Non-fluoroscopic Catheter Tracking for Fluoroscopy Reduction in Interventional Electrophysiology
10:46

Non-fluoroscopic Catheter Tracking for Fluoroscopy Reduction in Interventional Electrophysiology

Published on: May 26, 2015

14.0K
Robotic Ablation of Atrial Fibrillation
11:21

Robotic Ablation of Atrial Fibrillation

Published on: May 29, 2015

20.4K
Remote Magnetic Navigation for Accurate, Real-time Catheter Positioning and Ablation in Cardiac Electrophysiology Procedures
09:13

Remote Magnetic Navigation for Accurate, Real-time Catheter Positioning and Ablation in Cardiac Electrophysiology Procedures

Published on: April 21, 2013

28.7K

Area of Science:

  • Cardiology
  • Electrophysiology
  • Medical Complications

Background:

  • Slow pathway modification is a primary treatment for atrio-ventricular nodal re-entrant tachycardia (AVNRT).
  • Radiofrequency ablation (RFA) offers high success rates and low complication incidence for AVNRT.
  • Complete atrio-ventricular (AV) block is a rare complication of RFA.

Purpose of the Study:

  • To report a rare case of delayed complete heart block following RFA for AVNRT.
  • To highlight the importance of monitoring patients for delayed complications after AVNRT ablation.

Main Methods:

  • A case study of a patient undergoing RFA for AVNRT.
  • Observation of delayed complete heart block occurring 10 days post-procedure.
  • Monitoring of cardiac conduction until resolution.

Main Results:

  • The patient experienced delayed complete heart block 10 days after RFA for AVNRT.
  • Transient atrio-ventricular (AV) block during the procedure was noted.
  • Conduction returned to normal within 6 weeks.

Conclusions:

  • Delayed complete heart block is a rare but possible complication of RFA for AVNRT.
  • Patients experiencing transient AV block during RFA require close post-procedure monitoring for delayed complications.