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Related Concept Videos

Cranial and Spinal Meninges01:19

Cranial and Spinal Meninges

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The cranial and spinal meninges are complex protective structures surrounding the central nervous system (CNS), consisting of the brain and spinal cord. These meninges consist of the dura mater, the arachnoid mater, and the pia mater. They protect the CNS, provide structural support, and aid in circulating cerebrospinal fluid (CSF).
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The blood drainage from the head and neck is primarily managed by three pairs of veins: the external jugular, internal jugular, and vertebral veins. The external jugular veins drain superficial scalp and face structures, passing over the sternocleidomastoid muscles to empty into the subclavian veins.
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Related Experiment Video

Updated: Apr 17, 2026

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model
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Are acute subdural hematomas possible without head trauma?

D Garbossa1, R Altieri1, F M Calamo Specchia1

  • 1Department of Neurosurgery, University of Turin, Turin, Italy.

Asian Journal of Neurosurgery
|February 17, 2015
PubMed
Summary
This summary is machine-generated.

Acute subdural hematomas (ASDHs) are uncommon, especially after mild trauma. This study highlights coagulation deficits as a key risk factor, emphasizing the importance of coagulation and platelet function in managing ASDHs.

Keywords:
Acute subdural hematomasanticoagulant therapycoagulation deficit

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Area of Science:

  • Neurosurgery
  • Hematology
  • Trauma Medicine

Background:

  • Acute subdural hematomas (ASDHs) are rare neurological emergencies.
  • Mild head trauma is often insufficient to explain ASDH development.
  • Hypertension, vascular abnormalities, and coagulation deficits are identified risk factors.

Purpose of the Study:

  • To investigate the role of coagulation factors and platelets in patients with ASDH.
  • To review the literature concerning spontaneous ASDH and coagulation disorders.
  • To enhance understanding of ASDH management in patients with bleeding diatheses.

Main Methods:

  • Presentation of two clinical cases of ASDH in patients with diagnosed coagulation deficits.
  • Comprehensive review of existing medical literature on ASDH and coagulation abnormalities.
  • Analysis of the impact of coagulation factors and platelet function on ASDH outcomes.

Main Results:

  • Coagulation deficits were present in the reported ASDH cases.
  • Literature review supports the association between bleeding disorders and spontaneous ASDH.
  • Understanding the role of hemostasis is crucial for managing these rare conditions.

Conclusions:

  • Coagulation deficits represent a significant risk factor for developing acute subdural hematomas.
  • Proper management of ASDHs in patients with bleeding disorders requires attention to coagulation factors and platelet function.
  • Further research into the specific mechanisms linking coagulation to ASDH is warranted.