Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Cardiomyopathy II: Dilated Cardiomyopathy01:30

Cardiomyopathy II: Dilated Cardiomyopathy

803
Dilated cardiomyopathy, or DCM, is a progressive myocardial disorder characterized by ventricular chamber dilation and contractile dysfunction.EtiologyVarious factors can cause DCM, including hypertension and heavy alcohol intake, which contribute to the weakening and enlargement of the heart muscle. Viral infections, such as Coxsackievirus B, adenoviruses, and influenza, can lead to DCM by causing inflammation and damage to heart tissue. Certain chemotherapeutic agents, including daunorubicin,...
803
Cardiomyopathy V: Interprofessional Care01:29

Cardiomyopathy V: Interprofessional Care

711
Managing cardiomyopathy involves addressing underlying or precipitating causes, treating heart failure with medications, and implementing dietary changes and a balanced exercise and rest regimen.Lifestyle ModificationsCardiomyopathy patients should adopt a low-sodium diet to reduce fluid retention and manage heart failure. A personalized exercise and rest plan helps maintain physical fitness without overstraining the heart. Avoiding alcohol and tobacco is essential to prevent further damage to...
711
Mitral Regurgitation I: Introduction01:20

Mitral Regurgitation I: Introduction

1.2K
Mitral regurgitation is characterized by the backward circulation of blood from the left ventricle to the left atrium during systole, a phase of the cardiac cycle when the heart contracts and pumps blood out of the chambers. This abnormal flow occurs primarily due to the dysfunction of the mitral valve or its supporting structures, which include the mitral leaflets, chordae tendineae, annulus, and papillary muscles.Etiology and Mechanisms:Primary Mitral Regurgitation: This type arises from...
1.2K
Mitral Stenosis I: Introduction01:22

Mitral Stenosis I: Introduction

1.6K
Mitral Valve Stenosis (MVS) is a heart condition where the mitral valve narrows, impeding blood circulation from the left atrium to the left ventricle. The etiology and pathophysiology of this condition are multifaceted, leading to a cascade of cardiovascular complications.Causes of Mitral Valve StenosisRheumatic Heart Disease: It is the main cause of mitral valve stenosis, particularly in developing nations. This condition arises from rheumatic fever, an inflammatory illness resulting from...
1.6K
Cardiomyopathy III: Hypertrophic Cardiomyopathy01:29

Cardiomyopathy III: Hypertrophic Cardiomyopathy

793
Hypertrophic cardiomyopathy, or HCM, is an autosomal dominant genetic disorder characterized by asymmetric left ventricular hypertrophy without ventricular dilation. It is more common in men and is typically diagnosed in young, athletic adults.EtiologyHCM is primarily genetic and is caused by mutations in genes encoding sarcomeric proteins. Researchers have identified over 1400 mutations across at least 11 different genes. Among these, the most frequently occurring mutations are found in the...
793
Dysrhythmias IV: Characteristics of Bradyarrhythmias01:18

Dysrhythmias IV: Characteristics of Bradyarrhythmias

845
Bradyarrhythmias are cardiac rhythm disorders characterized by a slower-than-normal heart rate, typically defined as fewer than 60 beats per minute. Some of which are discussed here:Sinus BradycardiaSinus bradycardia presents a heart rate lower than 60 beats per minute, with a regular rhythm originating from the SA node. The ECG typically shows normal P waves preceding each QRS complex, a normal PR interval (0.12 to 0.20 seconds), and a normal QRS duration (0.06 to 0.10 seconds).First-Degree AV...
845

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Correction: Diagnostic accuracy and clinical impact of internist-performed point-of-care ultrasound in atrial fibrillation: a multicenter study.

Internal and emergency medicine·2026
Same author

Diagnostic accuracy and clinical impact of internist-performed point-of-care ultrasound in atrial fibrillation: a multicenter study.

Internal and emergency medicine·2026
Same author

Benefits of a comprehensive care model in patients with heart failure and anemia: UMIPIC program.

Revista clinica espanola·2026
Same author

Profiling Teen Social Networking Sites Users: Developmental, Identity, and Psychological Issues.

Clinical neuropsychiatry·2026
Same author

<i>Aspergillus</i> Endocarditis in Native Valves in Non-Traditional Hosts: A Systematic Review of a Case in a Patient with CREST Syndrome and Advanced Liver Cirrhosis.

Journal of fungi (Basel, Switzerland)·2025
Same author

Molecular Pathogenesis and Targeted Therapies in Eosinophilic Granulomatosis with Polyangiitis: An Updated Review.

International journal of molecular sciences·2025

Related Experiment Video

Updated: Apr 15, 2026

Implantation of the Syncardia Total Artificial Heart
16:11

Implantation of the Syncardia Total Artificial Heart

Published on: July 18, 2014

36.3K

[Right heart failure after pacemaker implantation].

Juan Gallego Galiana1, Genoveva López Castellanos1, Francesca Gioia1

  • 1Servicio de Medicina Interna, Hospital Ramón y Cajal, Madrid, España.

Medicina Clinica
|April 7, 2015
PubMed
Summary
This summary is machine-generated.

Severe tricuspid regurgitation (TR) caused by pacemaker (PM) cables is a rare cause of right heart failure (HF). Early diagnosis via advanced echocardiography is crucial for managing this condition, which is increasingly recognized.

Keywords:
EchocardiographyEcocardiografíaInsuficiencia cardiaca derechaInsuficiencia tricuspídeaMarcapasosPacemakerRight heart failureTricuspid regurgitation

More Related Videos

Implantation of Total Artificial Heart in Congenital Heart Disease
07:27

Implantation of Total Artificial Heart in Congenital Heart Disease

Published on: July 18, 2014

25.4K
Establishing a Swine Model of Post-myocardial Infarction Heart Failure for Stem Cell Treatment
08:24

Establishing a Swine Model of Post-myocardial Infarction Heart Failure for Stem Cell Treatment

Published on: May 25, 2020

7.6K

Related Experiment Videos

Last Updated: Apr 15, 2026

Implantation of the Syncardia Total Artificial Heart
16:11

Implantation of the Syncardia Total Artificial Heart

Published on: July 18, 2014

36.3K
Implantation of Total Artificial Heart in Congenital Heart Disease
07:27

Implantation of Total Artificial Heart in Congenital Heart Disease

Published on: July 18, 2014

25.4K
Establishing a Swine Model of Post-myocardial Infarction Heart Failure for Stem Cell Treatment
08:24

Establishing a Swine Model of Post-myocardial Infarction Heart Failure for Stem Cell Treatment

Published on: May 25, 2020

7.6K

Area of Science:

  • Cardiology
  • Medical Devices
  • Echocardiography

Background:

  • Pacemaker (PM) implantation can rarely lead to severe tricuspid regurgitation (TR).
  • This TR can be a cause of progressive right heart failure (HF), negatively impacting patient prognosis.
  • Understanding this complication is vital given the increasing prevalence of cardiac devices.

Observation:

  • Presents three clinical cases of right HF secondary to TR after PM implantation.
  • Right HF symptoms can manifest early or years after PM placement.
  • Diagnosis relies on echocardiography, with 3D and transesophageal modalities offering superior accuracy over 2D transthoracic views for PM-associated TR.

Findings:

  • Severe TR due to PM cable interference is a rare but significant cause of right heart failure.
  • Echocardiography, particularly 3D and transesophageal, is essential for accurate diagnosis.
  • Medical management is the primary treatment strategy due to high risks associated with other interventions.

Implications:

  • Increased awareness and improved diagnostic tools will likely lead to more frequent identification of this condition.
  • Early detection and appropriate management are key to improving outcomes for patients with PM-associated TR.
  • This highlights the importance of considering device-related complications in patients presenting with heart failure symptoms.