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[Experimental measles infection in rodents]

Voprosy Virusologii
|September 1, 1989
PubMed

Insights

Newborn hamsters are sensitive to measles virus strains L-16 and Lec. While acute meningoencephalitis and virus presence were observed, long-term measles virus persistence in hamster brains was not demonstrated.

Area of Science:

  • Virology
  • Neuroscience
  • Immunology

Context:

  • Measles virus (MeV) can cause severe neurological complications.
  • Subacute sclerosing panencephalitis (SSPE) is a rare, fatal complication of measles infection.
  • Animal models are crucial for studying MeV pathogenesis and persistence.

Purpose:

  • To investigate the susceptibility of newborn hamsters to different measles virus strains.
  • To determine the potential for measles virus persistence in the hamster brain.
  • To analyze the expression of measles virus proteins in infected hamster brains.

Summary:

  • Intracerebral inoculation of the L-16 MeV strain caused acute meningoencephalitis and death in 85-100% of newborn hamsters, with infectious virus, antigen, and genome detected in the brain for over 30 days.
  • The Lec strain, isolated from an SSPE patient, induced less severe disease and 40% mortality, with infectious virus detectable up to 20 days and genome up to 31 days post-inoculation.
  • All measles virus structural proteins, except hemagglutinin, were expressed in L-16 infected brains, while all were expressed in Lec-infected brains. Long-term persistence (90-180 days) of both strains was not observed.

Impact:

  • This study highlights the utility of the newborn hamster model for MeV neurovirulence research.
  • Findings contribute to understanding MeV neuropathogenesis and the factors influencing viral persistence.
  • Provides insights into host-virus interactions during measles virus infection of the central nervous system.

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