Management of catecholamine-induced stunned myocardium--a case report

Mohit Mittal1, M Radhakrishnan1, G S UmamaheswaraRao1

  • 1Department of Neuroanaesthesia, National Institute of Mental Health and Neurosciences, Bangalore, India.

Insights

Triple-H therapy for cerebral vasospasm can cause cardiac failure, which may be missed by monitoring blood pressure alone. Noninvasive cardiac output monitoring aided diagnosis and treatment, preventing neurological deficits.

Area of Science:

  • Neurology
  • Cardiology
  • Critical Care Medicine

Background:

  • Cerebral vasospasm is a common complication after subarachnoid hemorrhage.
  • Hypertensive, hypervolumic, and hemodilution (Triple-H) therapy is a standard treatment for symptomatic cerebral vasospasm.
  • Triple-H therapy can induce a hyperadrenergic state, potentially leading to cardiac dysfunction.

Observation:

  • A patient undergoing Triple-H therapy developed cardiac failure.
  • Cardiac dysfunction was not apparent when monitoring blood pressure alone.
  • Bioreactance noninvasive cardiac output monitoring was utilized for diagnosis.

Findings:

  • Continuous cardiac output monitoring identified cardiac failure during Triple-H therapy.
  • The patient's cardiac failure was successfully treated with milrinone and dobutamine.
  • The patient recovered without neurological deficits.

Implications:

  • Relying solely on blood pressure may lead to missed diagnoses of cardiac complications during Triple-H therapy.
  • Noninvasive cardiac output monitoring is a valuable tool for guiding therapy in patients receiving Triple-H therapy.
  • Early detection and management of cardiac dysfunction can prevent adverse neurological outcomes.

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