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Related Concept Videos

Gastritis-II: Pathophysiology01:17

Gastritis-II: Pathophysiology

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Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
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Gastritis-I: Introduction and Types01:27

Gastritis-I: Introduction and Types

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Gastritis, defined by the inflammation or irritation of the stomach lining or gastric mucosa, manifests in several distinct forms: acute, chronic, reactive, and a specific subtype known as autoimmune metaplastic atrophic gastritis.
Acute gastritis presents as a sudden inflammation triggered by various stressors to the stomach lining, such as exposure to corrosive agents, local irritants like aspirin and other NSAIDs, alcohol consumption, radiation therapy, physical trauma, severe burns, sepsis,...
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Gastritis III: Clinical Manifestations and Management01:23

Gastritis III: Clinical Manifestations and Management

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The clinical manifestations of gastritis can vary depending on the cause and type of gastritis, but some common symptoms may include the following.
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
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Pathophysiology of Peptic Ulcer Disease: Injurious Factors01:22

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Peptic ulcers are sores on the stomach's inner lining and the upper small intestine, which are the result of disruptions in the mucosal layer that houses parietal cells which produce gastric acid, and chief cells which secrete pepsinogen.
In the antrum region, G cells secrete the gastrin hormone that binds to gastrin-cholecystokinin-B (CCK2) receptors on parietal and enterochromaffin-like (ECL) cells in the fundic glands. Simultaneously, the vagus nerve releases acetylcholine, which binds...
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Other Disorders of Digestive System01:30

Other Disorders of Digestive System

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The gastrointestinal tract is susceptible to various disorders. If the lower esophageal sphincter is damaged, stomach acid can flow back into the esophagus, causing irritation and inflammation of the lining. This condition is called gastroesophageal reflux disease (known as heartburn) and may cause chest pain and difficulty swallowing. In the stomach, prolonged use of nonsteroidal anti-inflammatory drugs like aspirin, chronic alcohol consumption, bacterial infections such as Helicobacter...
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Mucosal Barrier of the Stomach01:25

Mucosal Barrier of the Stomach

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The gastric glands contain parietal cells that secrete hydrochloric acid (HCl) for digestion. The cells secrete HCl because it is highly corrosive and essential for breaking down food. To achieve this, they secrete hydrogen and chloride ions into the lumen of the gastric glands, which combine to form HCl.
Within parietal cells, carbonic acid is first formed through the reaction of water and carbon dioxide. The dissociation of carbonic acid releases bicarbonate and hydrogen ions. The bicarbonate...
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Related Experiment Video

Updated: Jan 2, 2026

Author Spotlight: Gastric Epithelial Cell Responses in Helicobacter pylori infection
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[Lymphocytic gastritis].

J Haot, A Jouret

    Acta Gastro-Enterologica Belgica
    |May 1, 1989
    PubMed
    Summary
    This summary is machine-generated.

    Lymphocytic gastritis, a distinct condition with high lymphocyte counts, often presents with weight loss and anorexia. This chronic condition may spontaneously resolve within two years.

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    Area of Science:

    • Gastroenterology
    • Histopathology
    • Immunology

    Context:

    • Lymphocytic gastritis is a newly identified histopathological entity.
    • Characterized by dense lymphocytic infiltration of gastric epithelium.
    • Distinct diagnostic criteria based on elevated lymphocyte counts.

    Purpose:

    • To describe the key features of lymphocytic gastritis.
    • To explore its clinical and endoscopic presentation.
    • To investigate its relationship with other gastritis types.

    Summary:

    • Lymphocytic gastritis shows dense lymphocytic infiltration, exceeding counts in other gastritis types.
    • Clinical signs include weight loss and anorexia; endoscopic findings involve enlarged rugae and aphthoid nodules.
    • It correlates with diffuse/corporeal varioliform gastritis but not antral varioliform gastritis.

    Impact:

    • Lymphocytic gastritis is a chronic condition with potential spontaneous remission.
    • Its etiology and pathogenesis remain unknown, but immunological factors are suspected.
    • Histological similarity to celiac disease suggests an autoimmune link.