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Prolapse of inverted ileal loops through a patent vitellointestinal duct
Ashish Pathak1, Nitin Agarwal2, Poonam Singh3
1Department of Pediatrics, RD Gardi Medical College, Ujjain, Madhya Pradesh, India Department of Public Health Sciences, Global Health (IHCAR), Stockholm, Solna, Sweden Department of Women and Children's Health, International Maternal and Child Health Unit, Uppsala University, Uppsala, Sweden.
Insights
A prolapsed patent vitellointestinal duct (PVID) in an infant presented as a Y-shaped umbilical lesion. Surgical correction was successful, highlighting PVID as a common symptomatic omphalomesenteric duct anomaly.
Area of Science:
- Pediatric Surgery
- Congenital Anomalies
- Gastrointestinal Surgery
Background:
- The omphalomesenteric duct (vitellointestinal duct) normally obliterates in fetal development.
- Failure of obliteration can lead to various congenital umbilical anomalies, including Meckel's diverticulum and patent vitellointestinal duct (PVID).
- PVID is the most common symptomatic anomaly arising from the patent omphalomesenteric duct.
Observation:
- A 2-month-old girl presented with a rapidly enlarging, polypoidal, Y-shaped umbilical lesion.
- The lesion was reddish, prolapsed, and discharged gaseous and fecal matter.
- Diagnosis was prolapsed inverted ileal loops through a patent vitellointestinal duct (PVID).
Findings:
- The patient had no associated congenital anomalies.
- Surgical management involved transumbilical exploration, wedge resection, and anastomosis.
- The child tolerated the surgery well with an uneventful postoperative recovery.
Implications:
- Prolapsed patent vitellointestinal duct (PVID) is a significant cause of symptomatic umbilical abnormalities in infants.
- Prompt surgical intervention is crucial to prevent complications associated with PVID.
- This case underscores the importance of recognizing and managing PVID in pediatric surgical practice.
Abstract:
We report a case of a prolapsed patent vitellointestinal duct (PVID) in a 2-month-old girl child who presented with sudden increase in size of a polypoidal lesion into a large, 'Y'-shaped reddish, prolapsing lesion, discharging gaseous and faecal matter at her umbilicus. The lesion was diagnosed as a prolapse of inverted ileal loops through the PVID. The child had no associated congenital anomalies. A transumbilical exploration was performed, followed by wedge resection and anastomosis. The child tolerated the procedure well and the postoperative course was uneventful. If the omphalomesenteric duct fails to obliterate a range of congenital defects related to the umbilicus, it can become clinically apparent. Meckel's diverticulum is the commonest of these defects but is most often asymptomatic. PVID is the most common symptomatic anomaly of the patent omphalomesenteric duct and requires prompt surgical correction to avoid complications.
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