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Binswanger's encephalopathy: a review.

C M Fisher1

  • 1Department of Neurology, Massachusetts General Hospital, Boston 02114.

Journal of Neurology
|February 1, 1989
PubMed
Summary

Binswanger's encephalopathy, a condition affecting white matter, is reviewed. Its causes, including high blood pressure and ischemia, are discussed, but a unifying hypothesis remains elusive.

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Area of Science:

  • Neurology
  • Neuroradiology
  • Pathology

Background:

  • Binswanger's encephalopathy is a subcortical arteriosclerotic encephalopathy.
  • It is characterized by demyelination and lacunar infarcts in the deep white matter.

Purpose of the Study:

  • To provide a comprehensive review of Binswanger's encephalopathy.
  • To discuss the pathogenesis, clinical presentation, and diagnostic modalities.

Main Methods:

  • Review of existing literature on Binswanger's encephalopathy.
  • Analysis of historical data, neuroimaging findings (CT, MRI), and pathological studies.

Main Results:

  • Binswanger's encephalopathy involves white matter hypomyelination, potentially linked to vascular factors.
  • Key contributing factors include hypertension, ischemia, hypoxia, and edema.
  • Differential diagnosis includes conditions like Alzheimer's disease and congophilic angiopathy.

Conclusions:

  • The pathogenesis of Binswanger's encephalopathy is multifactorial, involving vascular disease and ischemia.
  • A definitive unifying hypothesis for its development has not yet been established.
  • Further research is needed to fully elucidate the underlying mechanisms.

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