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  11. Pathogenesis And Epidemiology Of Chronic Pancreatitis.

Pathogenesis and epidemiology of chronic pancreatitis.

H Sarles1, J P Bernard, C Johnson

  • 1Service de Gastroenterologie, Hôpital Sainte Marguerite, Marseille, France.

Annual Review of Medicine
|January 1, 1989

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View abstract on PubMed

Summary
This summary is machine-generated.

Chronic pancreatitis, a common condition, involves pathological changes and is linked to a molecule called PSP. Research is advancing to understand its molecular biochemistry.

Area of Science:

  • Gastroenterology and Hepatology
  • Biochemistry
  • Pathology

Background:

  • Acute pancreatitis is a complication, not a cause, of chronic pancreatitis.
  • Chronic pancreatitis encompasses various forms with distinct causes but shared pathological alterations.
  • Chronic calcifying pancreatitis is the most prevalent type globally.

Purpose of the Study:

  • To describe different forms of chronic pancreatitis.
  • To highlight the role of PSP (a calcium stabilizer) in chronic calcifying pancreatitis.
  • To indicate the progression of chronic pancreatitis research into molecular biochemistry.

Main Methods:

  • Review of existing literature on chronic pancreatitis.
  • Description of pathological changes in chronic pancreatitis.

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  • Identification of key molecular players, such as PSP.

    • Main Results:

    • Chronic pancreatitis is distinct from acute pancreatitis, often presenting as a complication.
    • Chronic calcifying pancreatitis is characterized by lithiasis and involves a new family of molecules.
    • Pancreatic stone protein (PSP), a calcium stabilizer, is identified as a key factor.

    Conclusions:

    • Chronic pancreatitis research has advanced to the molecular biochemistry level.
    • PSP plays a significant role in the pathophysiology of chronic calcifying pancreatitis.
    • Understanding molecular mechanisms is crucial for future therapeutic strategies.