Somatic mutations of DICER1 and KMT2D are frequent in intraocular medulloepitheliomas

Felix Sahm1,2, Frederick A Jakobiec3, Jochen Meyer1,2

  • 1Clinical Cooperation Unit Neuropathology, German Cancer Research Center (DKFZ), and Department of Neuropathology University Hospital, Heidelberg, Germany.

Insights

This study identified recurrent DICER1 and KMT2D mutations in intraocular medulloepithelioma (IO-MEPL), a rare eye tumor. These genetic alterations suggest distinct molecular subtypes and potential therapeutic targets for IO-MEPL.

Area of Science:

  • Ophthalmology
  • Oncology
  • Genetics

Background:

  • Intraocular medulloepithelioma (IO-MEPL) is a rare embryonal neuroepithelial eye tumor with limited understanding of its molecular basis.
  • Cytogenetic and molecular pathogenesis of IO-MEPL remain largely unexplored.

Purpose of the Study:

  • To investigate the mutational landscape of IO-MEPL using targeted next-generation sequencing.
  • To identify key genes and molecular alterations driving IO-MEPL development.

Main Methods:

  • Targeted next-generation sequencing of 130 genes in 19 IO-MEPL samples.
  • Analysis of routinely prepared paraffin-embedded tumor tissues.
  • High-coverage genome sequencing on the Illumina NextSeq 500 platform.

Main Results:

  • Recurrent and mutually exclusive mutations in DICER1 (6 tumors) and KMT2D (5 tumors) were identified as significant molecular events in IO-MEPL.
  • Non-recurrent mutations in BRCA2, BRCA1, NOTCH2, CDH1, and GSE1 were also found.
  • DICER1-mutated IO-MEPL samples showed fewer chromosomal alterations and formed a distinct DNA methylation cluster.

Conclusions:

  • Recurrent somatic mutations in DICER1 and KMT2D are likely crucial in the molecular pathogenesis of sporadic IO-MEPL.
  • These findings suggest the existence of distinct molecular variants of IO-MEPL.
  • Further research into these mutations may lead to novel nonsurgical therapeutic strategies for IO-MEPL.

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