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Published on: July 6, 2022
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Simulated Ankle Equinus Affects Knee Kinematics During Gait.
Lisa C Drefus1, Jocelyn F Hafer2, David M Scher3
1Pediatric Rehabilitation, Hospital for Special Surgery, 535 East 70th Street, New York, NY 10021 USA ; Leon Root Motion Analysis Laboratory, Hospital for Special Surgery, 535 East 70th Street, New York, NY 10021 USA.
Summary
Simulated ankle equinus exceeding 10° caused increased knee flexion during gait initial contact. This suggests knee flexion may be a compensation strategy, impacting gait analysis and treatment planning for neuromuscular disorders.
Area of Science:
- Biomechanics
- Gait Analysis
- Orthopedics
Background:
- Distinguishing gait compensations from true abnormalities is crucial for effective intervention in neuromuscular disorders.
- Ankle equinus, a common gait deviation, can influence other joint kinematics.
- Understanding these relationships aids in targeted treatment planning.
Purpose of the Study:
- To investigate the impact of isolated ankle equinus on knee kinematics during the initial contact phase of gait.
- To determine the threshold of ankle equinus that elicits compensatory knee flexion.
Main Methods:
- Ten healthy subjects underwent 3D motion analysis under five conditions: shoe alone and with ankle foot orthosis simulating 10°, 20°, and 30° of plantar flexion.
- Repeated-measures ANOVA analyzed differences in knee flexion at initial contact across conditions.
- Primary outcome measure was knee flexion angle at initial contact.
Main Results:
- Simulated ankle equinus greater than 10° consistently resulted in increased knee flexion at initial contact.
- Observed knee flexion ranged from 7° to 22° with simulated equinus.
- A clear pattern of increased knee flexion was identified as a compensation for simulated ankle equinus.
Conclusions:
- Increased knee flexion at initial contact appears to be a compensatory strategy for ankle equinus exceeding 10°.
- For ankle equinus of 10° or less, observed knee flexion may indicate a true gait deviation.
- Findings have significant implications for cerebral palsy treatment, differentiating compensation from abnormality to optimize gait interventions.

