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Published on: November 4, 2021
Scorpion envenomation-induced acute thrombotic inferior myocardial infarction
Ahmet Oytun Baykan1, Mustafa Gür2, Armağan Acele3
1Department of Cardiology, Adana Numune Training and Research Hospital, Adana, Turkey. oytunbaykan@yahoo.com.
Insights
Scorpion stings can rarely cause serious cardiac events like myocardial infarction (MI). Prompt treatment with antivenom and acute coronary syndrome protocols is crucial for survival.
Area of Science:
- Cardiology
- Toxicology
- Emergency Medicine
Background:
- Serious cardiac emergencies are rare complications of scorpion envenomation, typically presenting as non-ST segment elevation myocardial infarction, cardiogenic shock, or myocarditis.
- Potential mechanisms involve sympathetic overstimulation, venom-induced catecholamine release, and direct myocardial toxicity leading to blood pressure dysregulation and coronary vasospasm.
Observation:
- A 55-year-old male presented with acute inferior wall myocardial infarction (MI) two hours post-scorpion sting.
- Coronary angiography identified a total thrombotic occlusion of the left circumflex artery.
Findings:
- Successful treatment involved glycoprotein IIb/IIIa inhibitors, thrombus aspiration, antivenom administration, and supportive care.
- This case highlights that life-threatening MI can be a complication of certain scorpion envenomations.
Implications:
- Scorpion envenomation should be considered a potential cause of acute coronary syndrome, especially in endemic areas.
- Management should include prompt recognition and treatment of myocardial infarction as an acute coronary syndrome.
- Further research into the specific venom components and their direct cardiac effects is warranted.
Abstract:
The occurrence of a serious cardiac emergency following scorpion envenomation has rarely been reported and, when so, mostly presented as non-ST segment elevation myocardial infarction, cardiogenic shock, or myocarditis. Possible mechanisms include imbalance in blood pressure and coronary vasospasm caused by the combination of sympathetic excitation, scorpion venom-induced release of catecholamines, and the direct effect of the toxin on the myocardium. We report a case of a 55-year-old man who presented with acute inferior wall myocardial infarction (MI) within 2 h of being stung by a scorpion. Coronary angiogram revealed total thrombotic occlusion of the left circumflex artery, which was treated successfully with glycoprotein IIb/IIIa inhibitor, thrombus aspiration, antivenom serum, and supportive therapy. Therefore, life-threatening MI can complicate the clinical course during some types of scorpion envenomation and should be managed as an acute coronary syndrome.
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