Related Experiment Videos
Factitious Ulcer Misdiagnosed as Pyoderma Gangrenosum
Elena Conde Montero1, Begoña Sánchez-Albisua1, Soledad Guisado2
1Hospital Infanta Leonor, Madrid, Spain.
Wounds : a Compendium of Clinical Research and Practice
|February 19, 2016
Summary
Dermatitis artefacta, a self-inflicted skin condition, is often misdiagnosed as pyoderma gangrenosum. Early suspicion and supportive care are crucial for accurate diagnosis and effective management of factitious ulcers.
Area of Science:
- Dermatology
- Psychosomatic Medicine
- Medical Case Reports
Background:
- Dermatitis artefacta presents a diagnostic challenge due to the patient's denial of self-inflicted lesions.
- Misdiagnosis is common, often leading to confusion with conditions like pyoderma gangrenosum.
- Delayed or incorrect diagnosis results in unnecessary investigations and potentially harmful treatments.
Observation:
- A case of recurrent factitious abdominal ulcer initially treated as pyoderma gangrenosum is presented.
- The patient's lesion morphology, history, and treatment resistance were difficult to explain by other diagnoses.
- The importance of considering dermatitis artefacta in unexplained dermatological presentations is highlighted.
Findings:
- Suspecting dermatitis artefacta is essential when clinical presentation and treatment outcomes are incongruent with initial diagnoses.
- A supportive, non-confrontational approach is vital for patient rapport and accurate diagnosis.
- In-patient management and long-term follow-up can be effective in managing recurrent factitious ulcers.
Implications:
- Clinicians should maintain a high index of suspicion for dermatitis artefacta in challenging cases.
- Adopting a patient-centered, empathetic approach can improve diagnostic accuracy and therapeutic outcomes.
- Early recognition and appropriate management of dermatitis artefacta can prevent patient harm and reduce healthcare costs.
Related Concept Videos
Peptic Ulcer Disease I: Introduction
1.0K
Peptic Ulcer Disease (PUD) is characterized by mucosal excavation in the esophagus, stomach, pylorus, or duodenum. It can manifest as acute or chronic based on the extent and duration of mucosal involvement.
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
1.0K
Gastritis III: Clinical Manifestations and Management
1.6K
The clinical manifestations of gastritis can vary depending on the cause and type of gastritis, but some common symptoms may include the following.
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
1.6K
Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies
767
Peptic ulcer disease (PUD) presents with diverse symptoms depending on the location and severity of the ulcer. Clinical manifestations of peptic ulcer include dull pain and a burning sensation in the mid-epigastric region.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
767
Gastritis-II: Pathophysiology
1.7K
Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
1.7K
Peptic Ulcer
27
Peptic ulcers are erosive lesions of the gastric or duodenal lining, most commonly caused by Helicobacter pylori infection. This Gram-negative, helical bacterium has adapted to survive the stomach’s acidic environment by producing urease, which converts urea into ammonia and carbon dioxide. The ammonia neutralizes gastric acid in the bacterium’s immediate environment, allowing colonization of the gastric mucosa. H. pylori attaches to mucus-secreting epithelial cells, penetrates the...
27
Peptic Ulcer Disease II: Pathophysiology
2.6K
Peptic Ulcer Disease (PUD) is characterized by the development of ulcers in the stomach or duodenal mucosa. Its pathophysiology is complex, involving a balance between damaging and protective elements.
Damaging agents such as Helicobacter pylori, gastric acid, pepsin, and nonsteroidal anti-inflammatory drugs (NSAIDs) can weaken the mucosal defense, allowing hydrogen ions to infiltrate back and harm epithelial cells.
Damaging agents such as Helicobacter pylori, gastric acid, pepsin, and nonsteroidal anti-inflammatory drugs (NSAIDs) can weaken the mucosal defense, allowing hydrogen ions to infiltrate back and harm epithelial cells.
2.6K