MiR-138 Acts as a Tumor Suppressor by Targeting EZH2 and Enhances Cisplatin-Induced Apoptosis in Osteosarcoma Cells

Ziqiang Zhu1, Jinshan Tang2, Jianqiang Wang1

  • 1Department of Orthopaedics, the Second Affiliated Hospital of Xuzhou Medical College, Xuzhou, JiangSu Province, China.

Plos One
|March 29, 2016
PubMed

Insights

MicroRNA-138 (miR-138) is downregulated in osteosarcoma and inhibits tumor growth. Upregulating miR-138 enhances sensitivity to cisplatin chemotherapy by targeting EZH2, offering a potential therapeutic strategy.

Area of Science:

  • Oncology
  • Molecular Biology
  • Genetics

Background:

  • Chemotherapeutic insensitivity is a significant challenge in osteosarcoma treatment.
  • MicroRNAs (miRNAs) are increasingly recognized for their role in drug resistance.

Purpose of the Study:

  • To investigate the role of miR-138 in cisplatin chemoresistance in osteosarcoma.
  • To explore the underlying molecular mechanisms involving EZH2.

Main Methods:

  • Real-time PCR to quantify miR-138 expression in osteosarcoma tissues and cell lines.
  • Cell proliferation, invasion, migration, and apoptosis assays to assess malignant phenotype.
  • Western blot and luciferase assays to confirm EZH2 as a direct target of miR-138.

Main Results:

  • MiR-138 expression was significantly downregulated in osteosarcoma.
  • Overexpression of miR-138 suppressed osteosarcoma cell proliferation, migration, and invasion.
  • MiR-138 directly targets and downregulates EZH2 expression.
  • Elevated miR-138 expression increased osteosarcoma cell sensitivity to cisplatin by targeting EZH2.

Conclusions:

  • MiR-138 functions as a tumor suppressor in osteosarcoma.
  • MiR-138 enhances osteosarcoma cell chemosensitivity to cisplatin through EZH2.
  • MiR-138 holds potential as a therapeutic agent for osteosarcoma treatment.

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