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Published on: August 6, 2013
The self-medication hypothesis: Evidence from terrorism and cigarette accessibility
Michael F Pesko1, Christopher F Baum2
1Department of Healthcare Policy and Research, Weill Cornell Medical College, Cornell University, United States.
Short-term stress motivates smoking, but smoking does not alleviate stress. This study found stress increases smoking behavior, not the reverse, using advanced econometric models on a large dataset of current and former smokers.
Area of Science:
- Behavioral Economics
- Public Health
- Econometrics
Background:
- Short-term stress is a known motivator for smoking.
- The self-medication hypothesis suggests smoking reduces stress.
Purpose of the Study:
- To investigate the causal relationship between stress and smoking.
- To determine if smoking alleviates short-term stress (self-medication effect).
- To examine if stress motivates smoking (motivation effect).
Main Methods:
- Utilized single equation and system instrumental variable models.
- Employed data from 240,388 current and former smokers on self-reported smoking and stress.
- Instrumented short-term stress using temporal distance from September 11, 2001.
- Instrumented smoking using cigarette accessibility (price changes, state border proximity).
Main Results:
- Without accounting for endogeneity, smoking appeared associated with increased stress.
- After accounting for endogeneity, no evidence supported smoking reducing short-term stress.
- A consistent positive effect of short-term stress on smoking behavior was observed.
Conclusions:
- The self-medication hypothesis regarding stress reduction through smoking is not supported by this study.
- Short-term stress is a significant driver of smoking initiation or continuation.
- Endogeneity must be addressed to accurately understand the stress-smoking relationship.
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