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Updated: Mar 22, 2026

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Published on: October 14, 2025
Hydrocortisone-induced anti-inflammatory effects in immature human enterocytes depend on the timing of exposure
Samuli Rautava1, W Allan Walker2, Lei Lu3
1Department of Pediatrics, University of Turku and Turku University Hospital, Turku, Finland; Mucosal Immunology and Biology Research Center, Massachusetts General Hospital for Children, Boston, Massachusetts; and.
Insights
Hydrocortisone can reduce inflammation in the immature human gut, but its effectiveness against inflammatory insults like IL-1β and TNF-α depends on treatment timing. This finding is crucial for understanding corticosteroid use in infants.
Area of Science:
- Gastroenterology
- Neonatology
- Immunology
Background:
- Immature gut inflammation contributes to necrotizing enterocolitis (NEC) risk.
- Prenatal corticosteroids may lower NEC, but postnatal dexamethasone has neurodevelopmental risks.
- Understanding hydrocortisone's direct effects on the infant gut is critical.
Purpose of the Study:
- To investigate hydrocortisone's role in gene expression and inflammation in immature human enterocytes.
- To determine the time-dependent effects of hydrocortisone on intestinal cells.
- To analyze hydrocortisone's impact on inflammatory responses to IL-1β and TNF-α.
Main Methods:
- Utilized cDNA microarray to analyze time-dependent hydrocortisone effects on H4 cells.
- Conducted fetal intestinal organ and cell culture experiments.
- Induced inflammatory responses with IL-1β and TNF-α, measuring IL-8 and IL-6 secretion.
Main Results:
- Hydrocortisone altered gene expression patterns in a time-dependent manner.
- Significantly reduced IL-1β-induced IL-8 and IL-6 secretion in fetal ileum and H4 cells.
- Inhibited IL-8 secretion in response to TNF-α when given concurrently, but not when given 48h prior.
Conclusions:
- Hydrocortisone attenuates IL-1β-induced inflammation in the immature gut.
- The timing of hydrocortisone administration influences its anti-inflammatory efficacy.
- Provides a basis for understanding differential corticosteroid effects in neonates.
Abstract:
The immature human gut has a propensity to exaggerated inflammatory responses that are thought to play a role in the pathogenesis of necrotizing enterocolitis (NEC). Prenatal exposure to corticosteroids has been reported to reduce the risk of NEC, while postnatal dexamethasone treatment is associated with adverse neurodevelopmental outcomes in preterm infants. The aim of this study was to investigate the direct role of hydrocortisone in gene expression patterns and inflammatory responses in immature human enterocytes. Time-dependent hydrocortisone effects in nontransformed primary human fetal intestinal epithelial cell line H4 were investigated by cDNA microarray. Fetal intestinal organ culture and cell culture experiments were conducted. Inflammatory responses were induced by stimulation with IL-1β and TNF-α with and without hydrocortisone. IL-8 and IL-6 expression and secretion were measured as functional readout. Here we report time-dependent hydrocortisone-induced changes in gene expression patterns detected by cDNA microarray. Hydrocortisone significantly attenuated IL-1β-induced inflammatory responses in the immature human gut when administered at the time of the proinflammatory insult: IL-1β-induced IL-8 and IL-6 secretion in the fetal ileum as well as H4 cells were significantly reduced. Hydrocortisone also inhibited IL-8 secretion in response to TNF-α. In contrast, TNF-α-induced IL-8 secretion was not reduced in cells treated with hydrocortisone for 48 h before stimulation. Our observations provide a physiological basis for understanding the differential clinical effects of corticosteroids in the immature human gut depending on the timing of treatment.
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