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Related Concept Videos

Homeostatic Imbalances in Body Temperature01:19

Homeostatic Imbalances in Body Temperature

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Hyperthermia occurs when the body's temperature becomes unusually high, often due to heat exposure, intense physical activity, or certain illnesses. This condition can create a dangerous cycle where elevated body temperature increases the metabolic rate, generating more heat and potentially leading to organ failure and brain damage. A severe form of hyperthermia, called heat stroke, can raise body temperature to life-threatening levels. Fever, on the other hand, is a controlled form of...
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Methods of reducing fever01:22

Methods of reducing fever

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The signs and symptoms of fever include hot and dry skin, flushed face, thirst, muscle aches, anorexia, headache, tachycardia, tachypnea, and fatigue. Elevated body temperature is reduced using two methods: pharmacological and nonpharmacological. Proper identification and treatment of the root cause of a fever is of utmost importance.
Pharmacological Methods of Reducing Fever:
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Increased Body Temperature01:25

Increased Body Temperature

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A body temperature above  38°C  (100.4 °F) is known as fever or pyrexia, and a person with fever is termed 'febrile.' Typically, the hypothalamus, a part of the brain that acts as the body's thermostat, regulates body temperature through a thermoregulatory setpoint. It receives signals from cold and warm thermal receptors throughout the body and adjusts the body's temperature accordingly. Fever occurs when this hypothalamic setpoint is altered, usually in...
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Decreased Body Temperature01:29

Decreased Body Temperature

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A decreased body temperature can occur in patients with hypothermia and frostbite. Heat loss with extended cold exposure overpowers the body's ability to create heat, resulting in hypothermia. Core temperature readings help classify hypothermia. Mild hypothermia is temperatures between 32 °C (89.6 °F) and 35°C (95 °F) and is caused by impaired thermoregulation. Moderate hypothermia is temperatures between 28 C (82.4 °F) and 32 °C (89.6 °F) caused by...
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Veins01:17

Veins

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Veins are an integral part of our circulatory system, serving as the blood vessels that transport blood from all body regions to the heart. They are a network of hollow tubes that carry blood low in oxygen from the body's cells back to the heart for reoxygenation. Veins are crucial for maintaining the body's overall fluid balance and the continuous circulation of blood.
Structure of Veins:
The structure of veins is specifically designed to assist in the low-pressure transportation of...
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Types of Fever01:25

Types of Fever

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Fever can be triggered by several factors, including infections, nervous system disorders, certain cancers, blood diseases like leukemia, embolism, thrombosis, heatstroke, dehydration, surgical trauma, crushing injuries, and allergic reactions.
Here are the different types of fever:
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Updated: Mar 21, 2026

A Preclinical Model of Exertional Heat Stroke in Mice
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Syncope on a Hot Summer Day.

David Luke Glancy1

  • 1Section of Cardiology, Department of Medicine, Louisiana State University Health Sciences Center New Orleans, New Orleans, Louisiana.

The American Journal of Cardiology
|May 2, 2016
PubMed
Summary
This summary is machine-generated.

Ventricular tachycardia and myocardial infarction can signal severe triple vessel coronary disease. This occurs even without evidence of a recent blockage in the arteries.

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Area of Science:

  • Cardiology
  • Internal Medicine
  • Clinical Science

Background:

  • Coronary artery disease (CAD) is a leading cause of mortality worldwide.
  • Typical presentations include angina, myocardial infarction, and heart failure.
  • Atypical presentations can pose diagnostic challenges.

Observation:

  • A 63-year-old male presented with recurrent syncope and myocardial infarction.
  • Electrophysiological studies revealed ventricular tachycardia as the cause of symptoms.
  • Coronary angiography showed significant triple vessel coronary disease.

Findings:

  • The patient's symptoms were attributed to ventricular tachycardia secondary to severe, non-obstructive triple vessel coronary disease.
  • There was no arteriographic evidence of acute plaque rupture or fresh occlusion.
  • This suggests that stable, severe coronary artery disease can precipitate life-threatening arrhythmias.

Implications:

  • Clinicians should consider severe coronary artery disease in patients with unexplained ventricular tachycardia and syncope, even without acute coronary syndrome.
  • This case highlights the importance of comprehensive cardiac evaluation in managing complex arrhythmias.
  • Further research may explore the mechanisms linking stable CAD to ventricular arrhythmias.