[The status of the adaptation system and hemodynamics in patients with chronic post-embolic pulmonary hypertension]
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Updated: Oct 16, 2026

Invasive Hemodynamic Monitoring of Aortic and Pulmonary Artery Hemodynamics in a Large Animal Model of ARDS
Published on: November 26, 2018
Abstract:
The article discusses the results of biochemical, hemodynamic, and angiographic studies in 33 patients with chronic post-embolic pulmonary hypertension of various degree. The authors show that prolonged occlusion of the pulmono-vascular channel causes predominant activity of the sympathoadrenal, hypophyseal-adrenal, and renin-angiotensin-aldosterone systems, which maintains and aggravates pulmonary hypertension. The degree and character of these changes are directly dependent on the volume of the affection of the pulmonary vascular channel.
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